Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses (Q28487342)

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English	Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses	scientific journal article

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24 January 2017

Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses (English)

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24 January 2017

Mycobacterium tuberculosis

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author name string

Buka Samten

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1

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24 January 2017

Xisheng Wang

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2

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24 January 2017

Peter F. Barnes

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3

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24 January 2017

1 December 2009

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24 January 2017

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24 January 2017

89 Suppl 1

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24 January 2017

S74–76

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24 January 2017

Evidence for occurrence of the ESAT-6 protein in Mycobacterium tuberculosis and virulent Mycobacterium bovis and for its absence in Mycobacterium bovis BCG.

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A Mycobacterium tuberculosis operon encoding ESAT-6 and a novel low-molecular-mass culture filtrate protein (CFP-10)

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ESAT-6 proteins: protective antigens and virulence factors?

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Systematic review: T-cell-based assays for the diagnosis of latent tuberculosis infection: an update

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Molecular analysis of genetic differences between Mycobacterium bovis BCG and virulent M. bovis

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Deciphering the biology of Mycobacterium tuberculosis from the complete genome sequence

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Conclusive evidence that the major T-cell antigens of the Mycobacterium tuberculosis complex ESAT-6 and CFP-10 form a tight, 1:1 complex and characterization of the structural properties of ESAT-6, CFP-10, and the ESAT-6*CFP-10 complex. Implications

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Individual RD1-region genes are required for export of ESAT-6/CFP-10 and for virulence of Mycobacterium tuberculosis

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The primary mechanism of attenuation of bacillus Calmette-Guerin is a loss of secreted lytic function required for invasion of lung interstitial tissue

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Acute infection and macrophage subversion by Mycobacterium tuberculosis require a specialized secretion system

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The Type I IFN response to infection with Mycobacterium tuberculosis requires ESX-1-mediated secretion and contributes to pathogenesis

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Direct extracellular interaction between the early secreted antigen ESAT-6 of Mycobacterium tuberculosis and TLR2 inhibits TLR signaling in macrophages

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ESAT-6 inhibits production of IFN-gamma by Mycobacterium tuberculosis-responsive human T cells

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Cyclic AMP response element-binding protein positively regulates production of IFN-gamma by T cells in response to a microbial pathogen

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CREB, ATF, and AP-1 transcription factors regulate IFN-gamma secretion by human T cells in response to mycobacterial antigen

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Complex complexes: signaling at the TCR.

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Identifiers

DOI

10.1016/S1472-9792(09)70017-4

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1935939

OpenCitations bibliographic resource ID

1935939

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1935939

Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses (Q28487342)

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Mycobacterium tuberculosis ESX-1 system-secreted protein ESAT-6 but not CFP10 inhibits human T-cell immune responses (Q28487342)

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