Plzf mediates transcriptional repression of HoxD gene expression through chromatin remodeling (Q28212389)

8

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Taha Merghoub

2

object named as

Taha Merghoub

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author name string

Maria Barna

1

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José A Costoya

3

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Davide Ruggero

4

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Matthew Branford

5

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Anna Bergia

6

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Bruno Samori

7

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language of work or name

English

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publication date

October 2002

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number of pages

12

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Transformation of axial skeleton due to overexpression of bmi-1 in transgenic mice

inferred from page(s)

published in

Developmental Cell

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volume

3

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page(s)

499-510

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issue

4

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cites work

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7 January 2021

The POZ domain: a conserved protein-protein interaction motif

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Plzf regulates limb and axial skeletal patterning

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Transgenic analysis of a potential Hoxd-11 limb regulatory element present in tetrapods and fish.

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7 January 2021

Altered retinoic acid sensitivity and temporal expression of Hox genes in polycomb-M33-deficient mice

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7 January 2021

Histone deacetylase associated with mSin3A mediates repression by the acute promyelocytic leukemia-associated PLZF protein

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Coordinate expression of the murine Hox-5 complex homoeobox-containing genes during limb pattern formation

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7 January 2021

Amino-terminal protein-protein interaction motif (POZ-domain) is responsible for activities of the promyelocytic leukemia zinc finger-retinoic acid receptor-alpha fusion protein

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The vertebrate limb: a model system to study the Hox/HOM gene network during development and evolution

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Structure and activity of regulatory elements involved in the activation of the Hoxd-11 gene during late gastrulation.

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In vivo targeted mutagenesis of a regulatory element required for positioning the Hoxd-11 and Hoxd-10 expression boundaries

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7 January 2021

Fusion proteins of the retinoic acid receptor-alpha recruit histone deacetylase in promyelocytic leukaemia

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7 January 2021

Importance of globin gene order for correct developmental expression.

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Distinct interactions of PML-RARalpha and PLZF-RARalpha with co-repressors determine differential responses to RA in APL.

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Two critical hits for promyelocytic leukemia

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SMRT corepressor interacts with PLZF and with the PML-retinoic acid receptor alpha (RARalpha) and PLZF-RARalpha oncoproteins associated with acute promyelocytic leukemia

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Molecular models for vertebrate limb development

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Transcriptional activation of Drosophila homeotic genes from distant regulatory elements

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Breaking colinearity in the mouse HoxD complex.

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Control of colinearity in AbdB genes of the mouse HoxD complex.

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Class II histone deacetylases are directly recruited by BCL6 transcriptional repressor

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Sequence-specific DNA binding and transcriptional regulation by the promyelocytic leukemia zinc finger protein.

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Role of the histone deacetylase complex in acute promyelocytic leukaemia

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Oncogenic transcription factors in the human acute leukemias

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Oligomerization of RAR and AML1 transcription factors as a novel mechanism of oncogenic activation.

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Quantitative analysis of the transcription factor AP2 binding to DNA by atomic force microscopy

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Imprinting a determined state into the chromatin of Drosophila

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Polycombing the genome: PcG, trxG, and chromatin silencing

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Retinoic acid (RA) and As2O3 treatment in transgenic models of acute promyelocytic leukemia (APL) unravel the distinct nature of the leukemogenic process induced by the PML-RARalpha and PLZF-RARalpha oncoproteins

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Analysis of the molecular genetics of acute promyelocytic leukemia in mouse models

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Scanning force microscopy of DNA deposited onto mica: equilibration versus kinetic trapping studied by statistical polymer chain analysis.

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The human polycomb group complex associates with pericentromeric heterochromatin to form a novel nuclear domain

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Locking in stable states of gene expression: transcriptional control during Drosophila development.

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The RAR alpha-PLZF chimera associated with Acute Promyelocytic Leukemia has retained a sequence-specific DNA-binding domain.

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7 January 2021

Gene transpositions in the HoxD complex reveal a hierarchy of regulatory controls.

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Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with a targeted deletion of the bmi-1 proto-oncogene

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7 January 2021

Mice doubly deficient for the Polycomb Group genes Mel18 and Bmi1 reveal synergy and requirement for maintenance but not initiation of Hox gene expression

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HOX4 genes encode transcription factors with potential auto- and cross-regulatory capacities

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A RA-dependent, tumour-growth suppressive transcription complex is the target of the PML-RARalpha and T18 oncoproteins

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Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

2718800

2718800

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference