WISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte death (Q24642477)

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English	WISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte death	scientific article

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25 January 2020

WISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte death (English)

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25 January 2020

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Kaliyamurthi Venkatachalam

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25 January 2020

Balachandar Venkatesan

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Anthony J Valente

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Peter C Melby

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Sailesh Nandish

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Robert A Clark

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Bysani Chandrasekar

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1 April 2009

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25 January 2020

Journal of Biological Chemistry

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14414-14427

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25 January 2020

Functions and mechanisms of action of CCN matricellular proteins

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Cellular and molecular mechanisms of fibrosis

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Cytotoxicity of TNFalpha is regulated by integrin-mediated matrix signaling

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WISP-1 attenuates p53-mediated apoptosis in response to DNA damage through activation of the Akt kinase

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WISP-1 is a Wnt-1- and beta-catenin-responsive oncogene

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TNF-R1 signaling: a beautiful pathway

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Transcriptional regulation by the phosphorylation-dependent factor CREB

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Transcription factors responsive to cAMP

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cAMP inhibits transforming growth factor-beta-stimulated collagen synthesis via inhibition of extracellular signal-regulated kinase 1/2 and Smad signaling in cardiac fibroblasts

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TGF-beta1 and WISP-1/CCN-4 can regulate each other's activity to cooperatively control osteoblast function

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Resveratrol inhibits high glucose-induced PI3K/Akt/ERK-dependent interleukin-17 expression in primary mouse cardiac fibroblasts

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TNF-mediated inflammatory disease.

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IL-17 stimulates MMP-1 expression in primary human cardiac fibroblasts via p38 MAPK- and ERK1/2-dependent C/EBP-beta , NF-kappaB, and AP-1 activation

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TNF provokes cardiomyocyte apoptosis and cardiac remodeling through activation of multiple cell death pathways

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Interleukin-17 stimulates C-reactive protein expression in hepatocytes and smooth muscle cells via p38 MAPK and ERK1/2-dependent NF-kappaB and C/EBPbeta activation

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Dynamic interactions between myocytes, fibroblasts, and extracellular matrix

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Left ventricular remodeling after myocardial infarction: past, present, and future

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Cardiac fibroblasts: friend or foe?

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Impact of brief oxidant stress on primary adult cardiac fibroblasts.

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cAMP-response element-binding protein mediates tumor necrosis factor-alpha-induced vascular smooth muscle cell migration

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21 January 2018

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1 reference

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21 January 2018

Regulation of CCAAT/Enhancer binding protein, interleukin-6, interleukin-6 receptor, and gp130 expression during myocardial ischemia/reperfusion

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

based on heuristic

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Assessment of myocardial injury by serum tumour necrosis factor alpha measurements in acute myocardial infarction

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

based on heuristic

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Pro-inflammatory Cytokines Stimulate Mitogen-activated Protein Kinase Subfamilies, Increase Phosphorylation of c-Jun and ATF2 and Upregulate c-Jun Protein in Neonatal Rat Ventricular Myocytes

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Tumor necrosis factor-alpha at acute myocardial infarction in rats and effects on cardiac fibroblasts

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Tissue expression and immunolocalization of tumor necrosis factor-alpha in postinfarction dysfunctional myocardium

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Interleukin-18-induced human coronary artery smooth muscle cell migration is dependent on NF-kappaB- and AP-1-mediated matrix metalloproteinase-9 expression and is inhibited by atorvastatin

1 reference

https://pubmed.ncbi.nlm.nih.gov/19339243

12 December 2020

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10.1074/JBC.M809757200

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25 January 2020

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2 references

Consolidated OpenCitations Corpus – April 2017

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Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19339243%20AND%20SRC:MED&resulttype=core&format=json

25 January 2020

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