gC1q receptor ligation selectively down-regulates human IL-12 production through activation of the phosphoinositide 3-kinase pathway (Q24336138)

positive regulation of protein kinase B signaling

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

Stephen N Waggoner

1

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Michael W Cruise

2

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Rachel Kassel

3

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Young S Hahn

Complement C1q regulates LPS-induced cytokine production in bone marrow-derived dendritic cells.

4

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language of work or name

English

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publication date

1 October 2005

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published in

Journal of Immunology

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volume

175

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page(s)

4706-14

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issue

7

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cites work

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Hepatitis C virus core protein inhibits interleukin 12 and nitric oxide production from activated macrophages

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C5a negatively regulates toll-like receptor 4-induced immune responses

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cC1q-R (calreticulin) and gC1q-R/p33: ubiquitously expressed multi-ligand binding cellular proteins involved in inflammation and infection

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Complement: a unique innate immune sensor for danger signals

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The relevance of complement to virus biology

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Regulation of interleukin-12 production in antigen-presenting cells

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Different cytokine profiles of peripheral blood mononuclear cells from patients with persistent and self-limited hepatitis C virus infection

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Subversion of immune responses by hepatitis C virus: immunomodulatory strategies beyond evasion?

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Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

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PI3K and negative regulation of TLR signaling.

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Complement component C1q induces endothelial cell adhesion and spreading through a docking/signaling partnership of C1q receptors and integrins

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gC1q-R/p33, a member of a new class of multifunctional and multicompartmental cellular proteins, is involved in inflammation and infection

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Repeat-induced gene silencing in mammals.

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The complement system in regulation of adaptive immunity

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PI3K-mediated negative feedback regulation of IL-12 production in DCs

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Interleukin-12 and the regulation of innate resistance and adaptive immunity

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Immunology of hepatitis B virus and hepatitis C virus infection

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Evidence for two distinct phosphatidylinositol kinases in fibroblasts. Implications for cellular regulation

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Wortmannin is a potent phosphatidylinositol 3-kinase inhibitor: the role of phosphatidylinositol 3,4,5-trisphosphate in neutrophil responses.

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Identification of a cellular protein specifically interacting with the precursor of the hepatitis B e antigen

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Epstein-Barr virus receptor of human B lymphocytes is the C3d receptor CR2

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Hepatitis C virus core selectively suppresses interleukin-12 synthesis in human macrophages by interfering with AP-1 activation

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gC1q-R/p32, a C1q-binding protein, is a receptor for the InlB invasion protein of Listeria monocytogenes

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IL-12 and IL-23: master regulators of innate and adaptive immunity.

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Staphylococcus aureus protein A recognizes platelet gC1qR/p33: a novel mechanism for staphylococcal interactions with platelets

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Legionella pneumophila suppresses interleukin-12 production by macrophages

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Hepatitis C virus core protein leads to immune suppression and liver damage in a transgenic murine model

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The Herpesvirus Saimiri open reading frame 73 gene product interacts with the cellular protein p32.

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Hepatitis C Virus Structural Proteins Impair Dendritic Cell Maturation and Inhibit In Vivo Induction of Cellular Immune Responses

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Inhibition of interferon-mediated antiviral activity by murine gammaherpesvirus 68 latency-associated M2 protein

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Antigen presentation and T cell stimulation by dendritic cells

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Interaction between complement receptor gC1qR and hepatitis C virus core protein inhibits T-lymphocyte proliferation

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Molecular mechanisms of endotoxin tolerance

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Suppression of Il-12 transcription in macrophages following Fc gamma receptor ligation

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Role of the phosphatidylinositol 3 kinase-Akt pathway in the regulation of IL-10 and IL-12 by Porphyromonas gingivalis lipopolysaccharide

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