Mutation of MSH3 in endometrial cancer and evidence for its functional role in heteroduplex repair (Q24318892)

GOA release 2020-03-11

DNA mismatch repair

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GOA release 2020-03-11

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4

A Berchuck

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author name string

J I Risinger

1

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A Umar

2

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J Boyd

3

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T A Kunkel

5

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J C Barrett

https://scigraph.springernature.com/pub.10.1038/ng0996-102

6

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language of work or name

English

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publication date

September 1996

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published in

Nature Genetics

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volume

14

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issue

1

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page(s)

102-5

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exact match

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cites work

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Destabilization of tracts of simple repetitive DNA in yeast by mutations affecting DNA mismatch repair

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A hPMS2 mutant cell line is defective in strand-specific mismatch repair

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Genomic instability in repeated sequences is an early somatic event in colorectal tumorigenesis that persists after transformation

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Genetic instability occurs in the majority of young patients with colorectal cancer

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Molecular mechanisms underlying hereditary nonpolyposis colorectal carcinoma

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Biochemistry and genetics of eukaryotic mismatch repair

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Mismatch repair gene defects in sporadic colorectal cancers with microsatellite instability

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Somatic mutations in the hMSH2 gene in microsatellite unstable colorectal carcinomas

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Mutations in the MSH3 gene preferentially lead to deletions within tracts of simple repetitive DNA in Saccharomyces cerevisiae

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Requirement of the yeast RTH1 5' to 3' exonuclease for the stability of simple repetitive DNA.

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Redundancy of Saccharomyces cerevisiae MSH3 and MSH6 in MSH2-dependent mismatch repair

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Requirement of the yeast MSH3 and MSH6 genes for MSH2-dependent genomic stability

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Mechanisms and biological effects of mismatch repair

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MSH6, a Saccharomyces cerevisiae protein that binds to mismatches as a heterodimer with MSH2.

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Mutations of GTBP in genetically unstable cells

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The yeast gene MSH3 defines a new class of eukaryotic MutS homologues.

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Normal human chromosome 11 suppresses tumorigenicity of human cervical tumor cell line SiHa

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DNA-replication fidelity, mismatch repair and genome instability in cancer cells

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Mismatch repair in replication fidelity, genetic recombination, and cancer biology

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GTBP, a 160-kilodalton protein essential for mismatch-binding activity in human cells

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Isolation of an hMSH2-p160 heterodimer that restores DNA mismatch repair to tumor cells

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An alkylation-tolerant, mutator human cell line is deficient in strand-specific mismatch repair.

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Male mice defective in the DNA mismatch repair gene PMS2 exhibit abnormal chromosome synapsis in meiosis

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Transcription-coupled repair deficiency and mutations in human mismatch repair genes

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Molecular cloning of the N-terminus of GTBP

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