The human Frizzled 6 (HFz6) acts as a negative regulator of the canonical Wnt. beta-catenin signaling cascade (Q24306497)

Frizzled class receptor 6

GOA release 2020-03-11

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negative regulation of canonical Wnt signaling pathway

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

Tamar Golan

1

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Abraham Yaniv

2

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Anna Bafico

3

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Guizhong Liu

4

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Arnona Gazit

Journal of Biological Chemistry

5

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language of work or name

English

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publication date

9 April 2004

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published in

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volume

279

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page(s)

14879-88

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issue

15

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cites work

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The TAK1-NLK mitogen-activated protein kinase cascade functions in the Wnt-5a/Ca(2+) pathway to antagonize Wnt/beta-catenin signaling

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Wnt-5/pipetail functions in vertebrate axis formation as a negative regulator of Wnt/beta-catenin activity.

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Increasingly complex: new players enter the Wnt signaling network

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An LDL-receptor-related protein mediates Wnt signalling in mice

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arrow encodes an LDL-receptor-related protein essential for Wingless signalling

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Biochemical interactions in the wnt pathway

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More than one way to skin a catenin

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Ca(2+)/calmodulin-dependent protein kinase II is stimulated by Wnt and Frizzled homologs and promotes ventral cell fates in Xenopus

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When Wnts antagonize Wnts.

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Regulation of lymphoid enhancer factor 1/T-cell factor by mitogen-activated protein kinase-related Nemo-like kinase-dependent phosphorylation in Wnt/beta-catenin signaling

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Human frizzled 1 interacts with transforming Wnts to transduce a TCF dependent transcriptional response

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Molecular cloning of human Frizzled-6

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A molecular mechanism for the effect of lithium on development

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Neoplastic transformation of RK3E by mutant beta-catenin requires deregulation of Tcf/Lef transcription but not activation of c-myc expression

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Beta-catenin mutations in cell lines established from human colorectal cancers

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The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway

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The androgen receptor can promote beta-catenin nuclear translocation independently of adenomatous polyposis coli

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Siah-1 mediates a novel beta-catenin degradation pathway linking p53 to the adenomatous polyposis coli protein

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Siah-1, SIP, and Ebi collaborate in a novel pathway for beta-catenin degradation linked to p53 responses

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Wnt-5a inhibits the canonical Wnt pathway by promoting GSK-3-independent beta-catenin degradation

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Interaction of Wnt and a Frizzled homologue triggers G-protein-linked phosphatidylinositol signalling

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Functional and structural diversity of the human Dickkopf gene family

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Identifiers

DOI

10.1074/JBC.M306421200

1 reference

Consolidated OpenCitations Corpus – April 2017

3163573

3163573

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference