Stimulation of glucose transport by AMP-activated protein kinase via activation of p38 mitogen-activated protein kinase (Q24291657)

positive regulation of MAP kinase activity

GOA release 2020-03-11

1 reference

Journal of Biological Chemistry

GOA release 2020-03-11

author name string

X Xi

series ordinal

1

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J Han

series ordinal

2

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J Z Zhang

series ordinal

3

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language of work or name

English

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publication date

2 November 2001

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published in

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volume

276

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page(s)

41029-34

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issue

44

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AMP-activated protein kinase: an ultrasensitive system for monitoring cellular energy charge.

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The regulation of AMP-activated protein kinase by phosphorylation

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Modulation of GLUT1 intrinsic activity in clone 9 cells by inhibition of oxidative phosphorylation

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Regulation of 5'-AMP-activated protein kinase activity by the noncatalytic beta and gamma subunits

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Characterization of the AMP-activated protein kinase kinase from rat liver and identification of threonine 172 as the major site at which it phosphorylates AMP-activated protein kinase

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Characterization of the structure and function of a novel MAP kinase kinase (MKK6)

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Dissociation of GLUT4 translocation and insulin-stimulated glucose transport in transgenic mice overexpressing GLUT1 in skeletal muscle

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Selective activation of p38 mitogen-activated protein (MAP) kinase isoforms by the MAP kinase kinases MKK3 and MKK6

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Specific inhibitors of p38 mitogen-activated protein kinase block 3T3-L1 adipogenesis

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Functional domains of the alpha1 catalytic subunit of the AMP-activated protein kinase

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An inhibitor of p38 mitogen-activated protein kinase prevents insulin-stimulated glucose transport but not glucose transporter translocation in 3T3-L1 adipocytes and L6 myotubes

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Molecular basis of insulin-stimulated GLUT4 vesicle trafficking. Location! Location! Location!

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Preconditioning enhanced glucose uptake is mediated by p38 MAP kinase not by phosphatidylinositol 3-kinase

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Effect of contraction on mitogen-activated protein kinase signal transduction in skeletal muscle. Involvement Of the mitogen- and stress-activated protein kinase 1

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MKK6/3 and p38 MAPK pathway activation is not necessary for insulin-induced glucose uptake but regulates glucose transporter expression

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Purification and characterization of the AMP-activated protein kinase. Copurification of acetyl-CoA carboxylase kinase and 3-hydroxy-3-methylglutaryl-CoA reductase kinase activities

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Facilitative glucose transporters

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5-aminoimidazole-4-carboxamide ribonucleoside. A specific method for activating AMP-activated protein kinase in intact cells?

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The AMP-activated protein kinase--fuel gauge of the mammalian cell?

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Evidence of two mechanisms for the activation of the glucose transporter GLUT1 by anisomycin: p38(MAP kinase) activation and protein synthesis inhibition in mammalian cells

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A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells

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The AMP-activated/SNF1 protein kinase subfamily: metabolic sensors of the eukaryotic cell?

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Enhancement of glucose transport in response to inhibition of oxidative metabolism: pre- and posttranslational mechanisms

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Overexpression of stomatin depresses GLUT-1 glucose transporter activity

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Mitogen-activated protein kinase: conservation of a three-kinase module from yeast to human

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Evidence for 5' AMP-activated protein kinase mediation of the effect of muscle contraction on glucose transport.

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5' AMP-activated protein kinase activation causes GLUT4 translocation in skeletal muscle

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5-aminoimidazole-4-carboxamide ribonucleoside (AICAR) inhibits insulin-stimulated glucose transport in 3T3-L1 adipocytes.

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Stimulation of MAPK cascades by insulin and osmotic shock: lack of an involvement of p38 mitogen-activated protein kinase in glucose transport in 3T3-L1 adipocytes

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Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport

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Constitutively active mitogen-activated protein kinase kinase increases GLUT1 expression and recruits both GLUT1 and GLUT4 at the cell surface in 3T3-L1 adipocytes

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Metabolic stress and altered glucose transport: activation of AMP-activated protein kinase as a unifying coupling mechanism

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Dissociation of AMP-activated protein kinase activation and glucose transport in contracting slow-twitch muscle

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