Differential ability of exogenous chemotactic agents to disrupt transendothelial migration of flowing neutrophils (Q22254128)

C-X-C motif chemokine ligand 8

1 reference

Complement C5

1 reference

C-X-C motif chemokine ligand 1

1 reference

C-X-C motif chemokine receptor 2

1 reference

tumor necrosis factor d1

1 reference

Formyl peptide receptor 1

1 reference

signal transduction

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Induction of neutrophil-mediated cartilage degradation by interleukin-8

1 reference

N T Luu

1

N T

Luu

1 reference

30 March 2023

G E Rainger

series ordinal

2

author given names

G E

author last names

Rainger

1 reference

30 March 2023

G B Nash

series ordinal

3

author given names

G B

author last names

Nash

1 reference

30 March 2023

language of work or name

English

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publication date

1 June 2000

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published in

Journal of Immunology

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volume

164

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page(s)

5961-9

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issue

11

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cites work

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The CXC Chemokines Growth-regulated Oncogene (GRO) α, GROβ, GROγ, Neutrophil-activating Peptide-2, and Epithelial Cell-derived Neutrophil-activating Peptide-78 Are Potent Agonists for the Type B, but Not the Type A, Human Interleukin-8 Receptor

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PAF mediates neutrophil adhesion to thrombin or TNF-stimulated endothelial cells under shear stress

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Kinetics of the different steps during neutrophil migration through cultured endothelial monolayers treated with tumour necrosis factor-alpha

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Interleukin-8 mediates interleukin-1 alpha-induced neutrophil transcellular migration

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Each step during transendothelial migration of flowing neutrophils is regulated by the stimulatory concentration of tumour necrosis factor-alpha

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