A novel function of VEGF receptor-2 (KDR): rapid release of nitric oxide in response to VEGF-A stimulation in endothelial cells (Q22010936)

positive regulation of nitric-oxide synthase biosynthetic process

GOA release 2020-03-11

1 reference

Biochemical and Biophysical Research Communications

GOA release 2020-03-11

J Kroll

1

J

Kroll

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7 February 2023

J Waltenberger

series ordinal

2

author given names

J

author last names

Waltenberger

1 reference

7 February 2023

language of work or name

English

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publication date

30 November 1999

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published in

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volume

265

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issue

3

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page(s)

636-9

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VEGF-A induces expression of eNOS and iNOS in endothelial cells via VEGF receptor-2 (KDR)

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Neuropilin-1 is expressed by endothelial and tumor cells as an isoform-specific receptor for vascular endothelial growth factor

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Placenta growth factor stimulates MAP kinase and mitogenicity but not phospholipase C-gamma and migration of endothelial cells expressing Flt 1

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The Vascular Endothelial Growth Factor Receptor Flt-1 Mediates Biological Activities

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The vascular endothelial growth factor receptor KDR activates multiple signal transduction pathways in porcine aortic endothelial cells

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Vascular endothelial growth factor signals endothelial cell production of nitric oxide and prostacyclin through flk-1/KDR activation of c-Src

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A novel vascular endothelial growth factor, VEGF-C, is a ligand for the Flt4 (VEGFR-3) and KDR (VEGFR-2) receptor tyrosine kinases

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A novel vascular endothelial growth factor encoded by Orf virus, VEGF-E, mediates angiogenesis via signalling through VEGFR-2 (KDR) but not VEGFR-1 (Flt-1) receptor tyrosine kinases

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VEGF upregulates ecNOS message, protein, and NO production in human endothelial cells

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Endothelial dysfunction in atherosclerosis.

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Vascular endothelial growth factor/vascular permeability factor enhances vascular permeability via nitric oxide and prostacyclin

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Vascular endothelial growth factor-induced endothelial cell migration and proliferation depend on a nitric oxide-mediated decrease in protein kinase Cdelta activity

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Nitric oxide synthase lies downstream from vascular endothelial growth factor-induced but not basic fibroblast growth factor-induced angiogenesis

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Nitric oxide synthase modulates angiogenesis in response to tissue ischemia

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The biology of vascular endothelial growth factor

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