Expansion of T helper type 17 lymphocytes in patients with chronic granulomatous disease (Q84938046)

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12 February 2020

title

Expansion of T helper type 17 lymphocytes in patients with chronic granulomatous disease (English)

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chronic granulomatous disease

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main subject

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3

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Anna Šedivá

6

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author name string

R Horváth

1

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D Rožková

2

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A Poloučková

4

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P Sedláček

5

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R Spíšek

7

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publication date

1 October 2011

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Clinical and Experimental Immunology

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published in

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volume

166

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issue

1

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page(s)

26-33

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Surface phenotype and antigenic specificity of human interleukin 17-producing T helper memory cells

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cites work

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12 December 2020

IL-23 and IL-17 in the establishment of protective pulmonary CD4+ T cell responses after vaccination and during Mycobacterium tuberculosis challenge

1 reference

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T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma

1 reference

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Impaired T(H)17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

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Induction and effector functions of T(H)17 cells.

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Deficiency of Th17 cells in hyper IgE syndrome due to mutations in STAT3

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Stimulation of dendritic cells via the dectin-1/Syk pathway allows priming of cytotoxic T-cell responses

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Chronic granulomatous disease as a risk factor for autoimmune disease

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A homozygous CARD9 mutation in a family with susceptibility to fungal infections

1 reference

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The dual nature of TH17 cells: shifting the focus to function

1 reference

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Development, cytokine profile and function of human interleukin 17-producing helper T cells

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Genetic, biochemical, and clinical features of chronic granulomatous disease.

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Microbial lipopeptides induce the production of IL-17 in Th cells.

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Requirement of interleukin 17 receptor signaling for lung CXC chemokine and granulocyte colony-stimulating factor expression, neutrophil recruitment, and host defense

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12 December 2020

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

1 reference

12 December 2020

Primary immunodeficiency diseases: dissectors of the immune system

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Increased expression of interleukin 17 in inflammatory bowel disease

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CD4+ T cells mediate abscess formation in intra-abdominal sepsis by an IL-17-dependent mechanism

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Requirement of interleukin-17A for systemic anti-Candida albicans host defense in mice

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Gastrointestinal involvement in chronic granulomatous disease

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Polyarthritis resembling juvenile rheumatoid arthritis in a girl with chronic granulomatous disease

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Glomerulonephritis associated with chronic granulomatous disease and systemic lupus erythematosus

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Mice with a disrupted IFN-gamma gene are susceptible to the induction of experimental autoimmune encephalomyelitis (EAE).

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Human interleukin-17: A T cell-derived proinflammatory cytokine produced by the rheumatoid synovium

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IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

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Phagocytosis of apoptotic neutrophils regulates granulopoiesis via IL-23 and IL-17.

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Transforming growth factor-beta induces development of the T(H)17 lineage

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Card9 controls a non-TLR signalling pathway for innate anti-fungal immunity

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The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

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The adaptor protein CARD9 is required for innate immune responses to intracellular pathogens

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Dectin-1 stimulation by Candida albicans yeast or zymosan triggers NFAT activation in macrophages and dendritic cells

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IL-21 initiates an alternative pathway to induce proinflammatory T(H)17 cells

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10.1111/J.1365-2249.2011.04449.X

Identifiers

DOI

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