Specific impairment of endothelium-derived hyperpolarizing factor-type relaxation in mesenteric arteries from streptozotocin-induced diabetic mice (Q83176839)

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scientific article published on 19 April 2006

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English	Specific impairment of endothelium-derived hyperpolarizing factor-type relaxation in mesenteric arteries from streptozotocin-induced diabetic mice	scientific article published on 19 April 2006

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19 January 2020

Specific impairment of endothelium-derived hyperpolarizing factor-type relaxation in mesenteric arteries from streptozotocin-induced diabetic mice (English)

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19 January 2020

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Takayuki Matsumoto

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19 January 2020

Kiyoto Miyamori

2

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19 January 2020

Tsuneo Kobayashi

3

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19 January 2020

Katsuo Kamata

4

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19 January 2020

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19 April 2006

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19 January 2020

Vascular Pharmacology

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19 January 2020

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19 January 2020

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19 January 2020

450-460

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19 January 2020

Endothelium-derived C-type natriuretic peptide: more than just a hyperpolarizing factor

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The endothelium-derived hyperpolarizing factor puzzle: a mechanism without a mediator?

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Endothelium-dependent hyperpolarization. Beyond nitric oxide and cyclic GMP.

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Endothelial dysfunction in diabetes.

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Endothelial dysfunction in the streptozotocin-induced diabetic apoE-deficient mouse

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Myoendothelial gap junctions may provide the pathway for EDHF in mouse mesenteric artery.

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7 January 2021

Endothelium-derived hyperpolarizing factor does not contribute to the decrease in endothelium-dependent relaxation in the aorta of streptozotocin-induced diabetic rats

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EDHF: new therapeutic targets?

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Role of endothelium-derived hyperpolarizing factor in endothelial dysfunction during diabetes.

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Inhibition of endothelium-dependent vascular relaxation by lysophosphatidylcholine: impact of lysophosphatidylcholine on mechanisms involving endothelium-derived nitric oxide and endothelium derived hyperpolarizing factor

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Evidence for selective inhibition by lysophosphatidylcholine of acetylcholine-induced endothelium-dependent hyperpolarization and relaxation in rat mesenteric artery

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7 January 2021

Alterations in endothelium-dependent hyperpolarization and relaxation in mesenteric arteries from streptozotocin-induced diabetic rats

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The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

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Kinase-dependent regulation of the intermediate conductance, calcium-dependent potassium channel, hIK1.

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Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?

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Contribution of connexins to the function of the vascular wall.

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Lysophosphatidylcholine inhibits bradykinin-induced phosphoinositide hydrolysis and calcium transients in cultured bovine aortic endothelial cells

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Changes in superoxide dismutase mRNA expression by streptozotocin-induced diabetes

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7 January 2021

Ca2+ mobilization in the aortic endothelium in streptozotocin-induced diabetic and cholesterol-fed mice

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7 January 2021

Impairment of endothelium-dependent relaxation and changes in levels of cyclic GMP in aorta from streptozotocin-induced diabetic rats

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7 January 2021

Preservation of endothelium-dependent relaxation in cholesterol-fed and streptozotocin-induced diabetic mice by the chronic administration of cholestyramine

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7 January 2021

Contractile responses in spontaneously diabetic mice. I. Involvement of superoxide anion in enhanced contractile response of aorta to norepinephrine in C57BL/KsJ(db/db) mice

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Effects of chronic administration of the novel endothelin antagonist J-104132 on endothelial dysfunction in streptozotocin-induced diabetic rat.

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7 January 2021

Relationship between peroxisome proliferator-activated receptors (PPAR alpha and PPAR gamma) and endothelium-dependent relaxation in streptozotocin-induced diabetic rats

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7 January 2021

Relationship among cholesterol, superoxide anion and endothelium-dependent relaxation in diabetic rats

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Effect of insulin treatment on smooth muscle contractility and endothelium-dependent relaxation in rat aortae from established STZ-induced diabetes

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IGF-I-induced enhancement of contractile response in organ-cultured aortae from diabetic rats is mediated by sustained thromboxane A2 release from endothelial cells

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Mechanisms underlying the chronic pravastatin treatment-induced improvement in the impaired endothelium-dependent aortic relaxation seen in streptozotocin-induced diabetic rats

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Impairment of endothelium-dependent arterial relaxation by lysolecithin in modified low-density lipoproteins

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Lysophosphatidylcholine inhibits surface receptor-mediated intracellular signals in endothelial cells by a pathway involving protein kinase C activation

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The gap junction communication channel

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High glucose induces alteration of gap junction permeability and phosphorylation of connexin-43 in cultured aortic smooth muscle cells

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10.1016/J.VPH.2006.02.007

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:16624628%20AND%20SRC:MED&resulttype=core&format=json

19 January 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:16624628%20AND%20SRC:MED&resulttype=core&format=json

19 January 2020

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