Glibenclamide, an antagonist of ATP sensitive K+ channels, blocks free radical generation in the rat myocardium (Q77700440)

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7 December 2019

title

Glibenclamide, an antagonist of ATP sensitive K+ channels, blocks free radical generation in the rat myocardium (English)

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7 December 2019

author name string

T Obata

series ordinal

1

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7 December 2019

Y Yamanaka

series ordinal

2

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7 December 2019

publication date

1 November 1998

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7 December 2019

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7 December 2019

volume

257

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7 December 2019

issue

1

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7 December 2019

page(s)

57-59

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ATP-regulated K+ channels protect the myocardium against ischemia/reperfusion damage

7 December 2019

cites work

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Sensitive assay of hydroxyl free radical formation utilizing high pressure liquid chromatography with electrochemical detection of phenol and salicylate hydroxylation products.

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Aromatic hydroxylation as a potential measure of hydroxyl-radical formation in vivo. Identification of hydroxylated derivatives of salicylate in human body fluids

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Hydroxylation of salicylate as an assay for hydroxyl radicals: a cautionary note

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7 January 2021

Activation of ATP-sensitive potassium channels: a novel pharmacological approach to myocardial protection?

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Arachidonic acid disrupts calcium dynamics in neonatal rat cardiac myocytes

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Prostaglandins and the release of the adrenergic transmitter

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Oxygen-derived free radicals in postischemic tissue injury

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In vivo monitoring of norepinephrine and .OH generation on myocardial ischemic injury by dialysis technique

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Intracranial microdialysis of salicylic acid to detect hydroxyl radical generation by monoamine oxidase inhibitor in the rat

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7 January 2021

Effect of iron (II) on the generation of hydroxyl free radicals in rat myocardium

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7 January 2021

Evidence of hydroxyl free radical generation by calcium overload in rat myocardium

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7 January 2021

Pharmacological evidence for the persistent activation of ATP-sensitive K+ channels in early phase of reperfusion and its protective role against myocardial stunning

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7 January 2021

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Identifiers

DOI

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7 December 2019

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