Bcl-2 expression by myeloid precursors in myelodysplastic syndromes: relation to disease progression (Q77143129)

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3 December 2019

title

Bcl-2 expression by myeloid precursors in myelodysplastic syndromes: relation to disease progression (English)

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3 December 2019

author name string

R E Davis

series ordinal

1

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3 December 2019

P L Greenberg

series ordinal

2

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3 December 2019

publication date

1 September 1998

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3 December 2019

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3 December 2019

volume

22

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3 December 2019

issue

9

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3 December 2019

page(s)

767-777

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Proposals for the classification of the myelodysplastic syndromes

3 December 2019

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Simultaneous assessment of cell kinetics and programmed cell death in bone marrow biopsies of myelodysplastics reveals extensive apoptosis as the probable basis for ineffective hematopoiesis

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Novel in situ double labeling for simultaneous detection of proliferation and apoptosis

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BCL2 protein is topographically restricted in tissues characterized by apoptotic cell death

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Bcl-2 gene promotes haemopoietic cell survival and cooperates with c-myc to immortalize pre-B cells

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Neither macromolecular synthesis nor myc is required for cell death via the mechanism that can be controlled by Bcl-2.

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bcl-2-immunoglobulin transgenic mice demonstrate extended B cell survival and follicular lymphoproliferation

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Progression from lymphoid hyperplasia to high-grade malignant lymphoma in mice transgenic for the t(14;18)

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Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

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Erythropoietin retards DNA breakdown and prevents programmed death in erythroid progenitor cells

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Haemopoietic colony stimulating factors promote cell survival by suppressing apoptosis

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Prognostic relevance of histological findings on bone marrow biopsy in myelodysplastic syndromes

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The relationship between bcl-2 expression and response to chemotherapy in acute leukaemia.

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Protooncogene expression in subpopulations of cells from leukemia patients

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In leukemic hematopoiesis CD34 antigen does not have the same significance as it does normal hematopoiesis.

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Karyotype in myelodysplastic syndromes: relations to morphology, clinical evolution, and survival

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Cytogenetic clonality analysis: typical patterns in myelodysplastic syndrome and acute myeloid leukaemia

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Bone marrow histology in myelodysplastic syndromes. I. Histological findings in myelodysplastic syndromes and comparison with bone marrow smears

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Bone marrow histology in myelodysplastic syndromes. II. Prognostic value of abnormal localization of immature precursors in MDS.

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Molecular cytogenetics and the myelodysplastic syndromes

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7 January 2021

bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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7 January 2021

Tumor suppressor p53 is a direct transcriptional activator of the human bax gene

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7 January 2021

Wild-type p53 induces apoptosis of myeloid leukaemic cells that is inhibited by interleukin-6

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Down-regulation of wild-type p53 activity interferes with apoptosis of IL-3-dependent hematopoietic cells following IL-3 withdrawal.

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Bcl-2 blocks p53-dependent apoptosis

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7 January 2021

10.1016/S0145-2126(98)00051-4

Identifiers

DOI

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3 December 2019

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