Defective CD4T cell priming and resistance to experimental autoimmune encephalomyelitis in TNF-deficient mice due to innate immune hypo-responsiveness (Q74590836)

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scientific article published on 01 October 2001

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English	Defective CD4T cell priming and resistance to experimental autoimmune encephalomyelitis in TNF-deficient mice due to innate immune hypo-responsiveness	scientific article published on 01 October 2001

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scholarly article

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11 November 2019

Defective CD4T cell priming and resistance to experimental autoimmune encephalomyelitis in TNF-deficient mice due to innate immune hypo-responsiveness (English)

1 reference

Europe PubMed Central

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11 November 2019

experimental autoimmune encephalomyelitis

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Giorgos Kollias

3

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11 November 2019

author name string

G Kassiotis

1

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11 November 2019

K Kranidioti

2

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11 November 2019

publication date

1 October 2001

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11 November 2019

Journal of Neuroimmunology

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11 November 2019

119

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11 November 2019

239-247

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11 November 2019

2

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11 November 2019

Tumor necrosis factor/cachectin increases permeability of endothelial cell monolayers by a mechanism involving regulatory G proteins

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Severity of symptoms and demyelination in MOG-induced EAE depends on TNFR1

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The instructive role of innate immunity in the acquired immune response

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The p55 TNF-alpha receptor plays a critical role in T cell alloreactivity

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TNF accelerates the onset but does not alter the incidence and severity of myelin basic protein-induced experimental autoimmune encephalomyelitis

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TNF modulates endothelial properties by decreasing cAMP

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Critical points of tumor necrosis factor action in central nervous system autoimmune inflammation defined by gene targeting

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Acute autoimmune encephalomyelitis in mice. II. Susceptibility is controlled by the combination of H-2 and histamine sensitization genes

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TNF is a potent anti-inflammatory cytokine in autoimmune-mediated demyelination.

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Innate immunity: impact on the adaptive immune response

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Physical mapping of the autoimmune disease susceptibility locus, Bphs: co-localization with a cluster of genes from the TNF receptor superfamily on mouse chromosome 6

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Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune re

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Locus controlling Bordetella pertussis-induced histamine sensitization (Bphs), an autoimmune disease-susceptibility gene, maps distal to T-cell receptor beta-chain gene on mouse chromosome 6.

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Identifiers

10.1016/S0165-5728(01)00403-9

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11585627%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:11585627%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

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