Reconstitution of hTERT restores tumorigenicity in melanoma-derived c-Myc low-expressing clones (Q74355235)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12082531%20AND%20SRC:MED&resulttype=core&format=json

10 November 2019

title

Reconstitution of hTERT restores tumorigenicity in melanoma-derived c-Myc low-expressing clones (English)

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10 November 2019

7

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10 November 2019

author name string

Annamaria Biroccio

1

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10 November 2019

Sarah Amodei

2

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10 November 2019

Barbara Benassi

3

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10 November 2019

Marco Scarsella

4

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10 November 2019

Anna Cianciulli

5

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10 November 2019

Marcella Mottolese

6

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10 November 2019

Carlo Leonetti

8

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10 November 2019

Gabriella Zupi

9

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10 November 2019

language of work or name

English

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publication date

1 May 2002

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10 November 2019

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10 November 2019

volume

21

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10 November 2019

issue

19

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10 November 2019

page(s)

3011-3019

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1205415

10 November 2019

exact match

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cites work

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c-Myc down-regulation increases susceptibility to cisplatin through reactive oxygen species-mediated apoptosis in M14 human melanoma cells.

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p53 deficiency rescues the adverse effects of telomere loss and cooperates with telomere dysfunction to accelerate carcinogenesis

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c-MYC induces mammary tumorigenesis by means of a preferred pathway involving spontaneous Kras2 mutations

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Human c-myc onc gene is located on the region of chromosome 8 that is translocated in Burkitt lymphoma cells

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The MYC protein activates transcription of the alpha-prothymosin gene.

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Telomerase activity in human leukemic cell lines is inhibited by antisense pentadecadeoxynucleotides targeted against c-myc mRNA.

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Telomerase-deficient mice with short telomeres are resistant to skin tumorigenesis.

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Increased epidermal tumors and increased skin wound healing in transgenic mice overexpressing the catalytic subunit of telomerase, mTERT, in basal keratinocytes.

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Myc-Max heterodimers activate a DEAD box gene and interact with multiple E box-related sites in vivo

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7 January 2021

Expression of mouse telomerase reverse transcriptase during development, differentiation and proliferation

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Short dysfunctional telomeres impair tumorigenesis in the INK4a(delta2/3) cancer-prone mouse

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7 January 2021

Telomerase reverse transcriptase gene is a direct target of c-Myc but is not functionally equivalent in cellular transformation.

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7 January 2021

Creation of human tumour cells with defined genetic elements

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7 January 2021

Inhibition of telomerase limits the growth of human cancer cells.

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7 January 2021

Telomeres shorten during ageing of human fibroblasts

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Inhibition of human telomerase in immortal human cells leads to progressive telomere shortening and cell death

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7 January 2021

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Telomerase expression in human somatic cells does not induce changes associated with a transformed phenotype

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7 January 2021

Extra c-myc oncogene copies in high risk cutaneous malignant melanoma and melanoma metastases

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Tumorigenic conversion of primary embryo fibroblasts requires at least two cooperating oncogenes

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A link between c-Myc-mediated transcriptional repression and neoplastic transformation

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7 January 2021

Myc represses transcription of the growth arrest gene gas1

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Antitumor effect of c-myc antisense phosphorothioate oligodeoxynucleotides on human melanoma cells in vitro and and in mice.

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7 January 2021

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hEST2, the putative human telomerase catalytic subunit gene, is up-regulated in tumor cells and during immortalization

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7 January 2021

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7 January 2021

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7 January 2021

Reconstitution of telomerase activity in normal human cells leads to elongation of telomeres and extended replicative life span

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7 January 2021

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7 January 2021

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7 January 2021

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