Coexpression of hepatocyte growth factor-Met: an early step in ovarian carcinogenesis? (Q73787127)

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6 November 2019

title

Coexpression of hepatocyte growth factor-Met: an early step in ovarian carcinogenesis? (English)

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6 November 2019

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7

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6 November 2019

author name string

Wong AS

1

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6 November 2019

Pelech SL

2

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6 November 2019

Woo MM

3

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6 November 2019

Yim G

4

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6 November 2019

Rosen B

5

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6 November 2019

Ehlen T

6

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6 November 2019

Auersperg N

8

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6 November 2019

language of work or name

English

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publication date

1 March 2001

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6 November 2019

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6 November 2019

volume

20

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6 November 2019

page(s)

1318-1328

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6 November 2019

issue

11

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1204253

6 November 2019

exact match

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Evidence that hepatocyte growth factor abrogates contact inhibition of mitosis in Madin-Darby canine kidney cell monolayers

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Identification of the Hepatocyte Growth Factor Receptor As the c- met Proto-Oncogene Product

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Hepatocyte growth factor/scatter factor induces a variety of tissue-specific morphogenic programs in epithelial cells

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Hepatocyte growth factor stimulates motility, chemotaxis and mitogenesis in ovarian carcinoma cells expressing high levels of c-met

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Overexpression of the Met/HGF receptor in ovarian cancer.

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Autonomy of the epithelial phenotype in human ovarian surface epithelium: changes with neoplastic progression and with a family history of ovarian cancer

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Opposing actions of hepatocyte growth factor and basic fibroblast growth factor on cell contact, intracellular free calcium levels, and rat ovarian surface epithelial cell viability

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Hepatocyte growth factor induces rat ovarian surface epithelial cell mitosis or apoptosis depending on the presence or absence of an extracellular matrix

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Comparison of c-met expression in ovarian epithelial tumors and normal epithelia of the female reproductive tract by quantitative laser scan microscopy

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Hepatocyte growth factor/scatter factor stimulates chemotaxis and growth of malignant mesothelioma cells through c-met receptor

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Reciprocal interactions between human ovarian surface epithelial cells and adjacent extracellular matrix

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Simian virus 40-transformed human ovarian surface epithelial cells escape normal growth controls but retain morphogenetic responses to extracellular matrix

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Activation of hepatocyte growth factor in the injured tissues is mediated by hepatocyte growth factor activator.

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Hepatocyte growth factor/scatter factor induces not only scattering but also cohort migration of human colorectal-adenocarcinoma cells

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Mesenchymal-epithelial interactions in the ovarian follicle involve keratinocyte and hepatocyte growth factor production by thecal cells and their action on granulosa cells

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Hepatocyte growth factor promotes growth and lumen formation of fetal lung epithelial cells in primary culture

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Scatter factor/hepatocyte growth factor and its receptor, the c-met tyrosine kinase, can mediate a signal exchange between mesenchyme and epithelia during mouse development

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