Caspase-mediated degradation of AMPA receptor subunits: a mechanism for preventing excitotoxic necrosis and ensuring apoptosis (Q73770804)

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6 November 2019

title

Caspase-mediated degradation of AMPA receptor subunits: a mechanism for preventing excitotoxic necrosis and ensuring apoptosis (English)

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6 November 2019

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4

M P Mattson

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6 November 2019

author name string

G W Glazner

series ordinal

1

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6 November 2019

S L Chan

series ordinal

2

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6 November 2019

C Lu

series ordinal

3

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6 November 2019

publication date

1 May 2000

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6 November 2019

published in

Journal of Neuroscience

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6 November 2019

volume

20

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6 November 2019

page(s)

3641-3649

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6 November 2019

issue

10

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Activation of a caspase 3-related cysteine protease is required for glutamate-mediated apoptosis of cultured cerebellar granule neurons

6 November 2019

cites work

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Combined mechanical trauma and metabolic impairment in vitro induces NMDA receptor-dependent neuronal cell death and caspase-3-dependent apoptosis.

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Simultaneous Degradation of αII- and βII-Spectrin by Caspase 3 (CPP32) in Apoptotic Cells

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A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis

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Increased vulnerability of hippocampal neurons to excitotoxic necrosis in presenilin-1 mutant knock-in mice.

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NGF and bFGF protect rat hippocampal and human cortical neurons against hypoglycemic damage by stabilizing calcium homeostasis

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Apoptosis induced via AMPA-selective glutamate receptors in cultured murine cortical neurons.

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Activation of membrane-associated procaspase-3 is regulated by Bcl-2.

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The dietary excitotoxins beta-N-methylamino-L-alanine and beta-N-oxalylamino-L-alanine induce necrotic- and apoptotic-like death of rat cerebellar granule cells.

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Basic fibroblast growth factor selectively increases AMPA-receptor subunit GluR1 protein level and differentially modulates Ca2+ responses to AMPA and NMDA in hippocampal neurons

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Evidence for synaptic apoptosis.

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ALS-linked Cu/Zn-SOD mutation increases vulnerability of motor neurons to excitotoxicity by a mechanism involving increased oxidative stress and perturbed calcium homeostasis

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A cloning method for caspase substrates that uses the yeast two-hybrid system: cloning of the antiapoptotic gene gelsolin

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

The C-terminal domain of glutamate receptor subunit 1 is a target for calpain-mediated proteolysis.

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Basic FGF regulates the expression of a functional 71 kDa NMDA receptor protein that mediates calcium influx and neurotoxicity in hippocampal neurons

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Alternative cleavage of Alzheimer-associated presenilins during apoptosis by a caspase-3 family protease

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Role of caspases in N-methyl-D-aspartate-induced apoptosis in cerebrocortical neurons.

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Fibroblast growth factor and glutamate: opposing roles in the generation and degeneration of hippocampal neuroarchitecture

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12 December 2020

10.1523/JNEUROSCI.20-10-03641.2000

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10804206%20AND%20SRC:MED&resulttype=core&format=json

6 November 2019

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6 November 2019

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