Enhancement of tamoxifen-induced E-cadherin function by Ca2+ channel antagonists in human breast cancer MCF7/6 cells (Q71979084)

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22 October 2019

title

Enhancement of tamoxifen-induced E-cadherin function by Ca2+ channel antagonists in human breast cancer MCF7/6 cells (English)

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22 October 2019

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C Charlier

1

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22 October 2019

E Bruyneel

2

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22 October 2019

C Lechanteur

3

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22 October 2019

M Bracke

4

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22 October 2019

M Mareel

5

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22 October 2019

V Castronovo

6

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22 October 2019

language of work or name

English

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publication date

1 December 1996

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European Journal of Pharmacology

22 October 2019

published in

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22 October 2019

volume

317

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22 October 2019

issue

2-3

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22 October 2019

page(s)

413-416

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Tamoxifen up-regulates c-erbB-2 expression in oestrogen-responsive breast cancer cells in vitro

22 October 2019

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In VitroInteraction of Estradiol Receptor with Ca2+-Calmodulin

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Tamoxifen and its active metabolite inhibit growth of estrogen receptor-negative MDA-MB-435 cells

7 January 2021

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A randomized clinical trial evaluating tamoxifen in the treatment of patients with node-negative breast cancer who have estrogen-receptor-positive tumors

7 January 2021

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Estrogen receptor mutations in breast cancer

7 January 2021

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Potentiation of tamoxifen activity by verapamil in a human breast cancer cell line

7 January 2021

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Regulation of the levels of three transforming growth factor beta mRNAs by estrogen and their effects on the proliferation of human breast cancer cells

7 January 2021

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Structural basis of cell-cell adhesion by cadherins

7 January 2021

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A human cell line from a pleural effusion derived from a breast carcinoma

7 January 2021

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A survey of human breast cancer sensitivity to growth inhibition by calmodulin antagonists in tissue culture

7 January 2021

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Tamoxifen inhibits lipid peroxidation in cardiac microsomes. Comparison with liver microsomes and potential relevance to the cardiovascular benefits associated with cancer prevention and treatment by tamoxifen

7 January 2021

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10.1016/S0014-2999(96)00816-3

7 January 2021

Identifiers

DOI

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22 October 2019

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