ICE inhibitor YVADcmk is a potent therapeutic agent against in vivo liver apoptosis (Q71527279)

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19 October 2019

title

ICE inhibitor YVADcmk is a potent therapeutic agent against in vivo liver apoptosis (English)

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19 October 2019

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5

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19 October 2019

author name string

N Rouquet

1

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19 October 2019

J C Pagès

2

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19 October 2019

T Molina

3

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19 October 2019

P Briand

4

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19 October 2019

publication date

1 September 1996

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19 October 2019

language of work or name

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19 October 2019

volume

6

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19 October 2019

issue

9

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19 October 2019

page(s)

1192-1195

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Involvement of the CD95 (APO-1/Fas) receptor and ligand in liver damage.

19 October 2019

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Fas- and tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene product

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Requirement of an ICE/CED-3 protease for Fas/APO-1-mediated apoptosis

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Involvement of an ICE-like protease in Fas-mediated apoptosis

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Induction of apoptosis by the mouse Nedd2 gene, which encodes a protein similar to the product of the Caenorhabditis elegans cell death gene ced-3 and the mammalian IL-1 beta-converting enzyme

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7 January 2021

Ich-1, an Ice/ced-3-related gene, encodes both positive and negative regulators of programmed cell death

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Identification and characterization of ICH-2, a novel member of the interleukin-1 beta-converting enzyme family of cysteine proteases

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Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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Yama/CPP32 beta, a mammalian homolog of CED-3, is a CrmA-inhibitable protease that cleaves the death substrate poly(ADP-ribose) polymerase

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A novel human protease similar to the interleukin-1 beta converting enzyme induces apoptosis in transfected cells

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Prevention of vertebrate neuronal death by the crmA gene

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Tumor necrosis factor-induced apoptosis is mediated by a CrmA-sensitive cell death pathway

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Fas-mediated apoptosis in primary cultured mouse hepatocytes

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7 January 2021

Bcl–2 protects from lethal hepatic apoptosis induced by an ant–Fas antibody in mice

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7 January 2021

The pathophysiology of tumor necrosis factors

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Lethal toxicity of lipopolysaccharide and tumor necrosis factor in normal and D-galactosamine-treated mice

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Anticytokine strategies in the treatment of the systemic inflammatory response syndrome

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Mice deficient for the 55 kd tumor necrosis factor receptor are resistant to endotoxic shock, yet succumb to L. monocytogenes infection

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Mice deficient in IL-1 beta-converting enzyme are defective in production of mature IL-1 beta and resistant to endotoxic shock

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7 January 2021

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Identifiers

DOI

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19 October 2019

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