Immunohistochemical detection of advanced glycation end products in dialysis-related amyloidosis (Q71517603)

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19 October 2019

title

Immunohistochemical detection of advanced glycation end products in dialysis-related amyloidosis (English)

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19 October 2019

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T Niwa

1

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19 October 2019

S Miyazaki

2

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19 October 2019

T Katsuzaki

3

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N Tatemichi

4

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Y Takei

5

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19 October 2019

T Miyazaki

6

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T Morita

7

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Y Hirasawa

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language of work or name

English

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publication date

1 September 1995

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19 October 2019

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19 October 2019

volume

48

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19 October 2019

issue

3

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19 October 2019

page(s)

771-778

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https://scigraph.springernature.com/pub.10.1038/ki.1995.349

19 October 2019

exact match

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cites work

A new form of amyloid protein associated with chronic hemodialysis was identified as beta 2-microglobulin

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Beta-2 microglobulin is an amyloidogenic protein in man.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Beta 2-microglobulin: a new form of amyloid protein associated with chronic hemodialysis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Polymerization of intact beta 2-microglobulin in tissue causes amyloidosis in patients on chronic hemodialysis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Serum levels of beta 2-microglobulin as a new form of amyloid protein in patients undergoing long-term hemodialysis.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

beta 2-Microglobulin modified with advanced glycation end products is a major component of hemodialysis-associated amyloidosis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Involvement of beta 2-microglobulin modified with advanced glycation end products in the pathogenesis of hemodialysis-associated amyloidosis. Induction of human monocyte chemotaxis and macrophage secretion of tumor necrosis factor-alpha and interleu

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

High-affinity-receptor-mediated uptake and degradation of glucose-modified proteins: a potential mechanism for the removal of senescent macromolecules

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Isolation of surface binding protein specific for advanced glycosylation end products from mouse macrophage-derived cell line RAW 264.7.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Nonenzymatic glycosylation and the pathogenesis of diabetic complications

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Accelerated age-related browning of human collagen in diabetes mellitus

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Hemoglobin-AGE: a circulating marker of advanced glycosylation

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Aging of proteins: isolation and identification of a fluorescent chromophore from the reaction of polypeptides with glucose

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Advanced glycation end products contribute to amyloidosis in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Advanced Maillard reaction end products are associated with Alzheimer disease pathology

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Detection of D-glucose-derived pyrrole compounds during Maillard reaction under physiological conditions

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Crosslines A and B as candidates for the fluorophores in age- and diabetes- related cross-linked proteins, and the diacetates produced by Maillard reaction of a-N-acetyl-L-lysine with D-glucose.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Evidence against the formation of 2-amino-6-(2-formyl-5-hydroxymethyl-pyrrol-l-yl)-hexanoic acid ('pyrraline') as an early-stage product or advanced glycation end product in non-enzymic protein glycation

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Continuous cultures of fused cells secreting antibody of predefined specificity

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Endothelial receptor-mediated binding of glucose-modified albumin is associated with increased monolayer permeability and modulation of cell surface coagulant properties

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Human and rat mesangial cell receptors for glucose-modified proteins: potential role in kidney tissue remodelling and diabetic nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Cachectin/TNF and IL-1 induced by glucose-modified proteins: role in normal tissue remodeling

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Macrophage/monocyte receptor for nonenzymatically glycosylated protein is upregulated by cachectin/tumor necrosis factor

1 reference

https://api.crossref.org/works/10.1038%2FKI.1995.349

7 January 2021

Identifiers

DOI

10.1038/KI.1995.349

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:7474663%20AND%20SRC:MED&resulttype=core&format=json

19 October 2019

1 reference