Evolution within the multigene family coding for the class I histocompatibility antigens: the case of the mouse t-haplotypes (Q70084649)

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8 October 2019

title

Evolution within the multigene family coding for the class I histocompatibility antigens: the case of the mouse t-haplotypes (English)

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8 October 2019

author name string

C Delarbre

1

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8 October 2019

T Morita

2

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8 October 2019

P Kourilsky

3

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8 October 2019

G Gachelin

4

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8 October 2019

publication date

1 January 1985

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8 October 2019

volume

136C

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8 October 2019

issue

1

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8 October 2019

page(s)

51-70

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Gene mapping within the T/t complex of the mouse. I. t-Lethal genes are nonallelic

8 October 2019

cites work

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Gene mapping within the T/t complex of the mouse. II. Anomalous position of the H-2 complex in t haplotypes

1 reference

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7 January 2021

Structure and expression of a mouse major histocompatibility antigen gene, H-2Ld

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7 January 2021

Identification of the class I genes of the mouse major histocompatibility complex by DNA-mediated gene transfer

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7 January 2021

Unusual evolutionary conservation and frequent DNA segment exchange in class I genes of the major histocompatibility complex

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Structure of class I major histocompatibility antigens.

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Lack of association between intrachromosomal gene conversion and reciprocal exchange

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7 January 2021

Polymorphism of the mouse H-2 loci

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7 January 2021

Genetics of the major histocompatibility complex: the final act.

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7 January 2021

The Control of Differentiation by the T Complex

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7 January 2021

Alternative RNA splicing in expression of the H-2K gene.

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7 January 2021

Different exon-intron organization at the 5' part of a mouse class I gene is used to generate a novel H-2Kd-related mRNA

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7 January 2021

A cDNA clone containing the entire coding sequence of a mouse H-2Kd histocompatibility antigen

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7 January 2021

Transmission ratio distortion in mouse t-haplotypes is due to multiple distorter genes acting on a responder locus

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7 January 2021

A nonpolymorphic class I gene in the murine major histocompatibility complex

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A potential donor gene for the bm1 gene conversion event in the C57BL mouse

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DNA sequence of a gene encoding a BALB/c mouse Ld transplantation antigen.

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7 January 2021

An H-2K gene of the tw32 mutant at the T/t complex is a close parent of an H-2Kq gene

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7 January 2021

Evolutionary relationships between the t and H-2 haplotypes in the house mouse

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7 January 2021

Biochemical studies on the H-2K mutant B6.C-H-2bm10

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7 January 2021

A major rearrangement in the H-2 complex of mouse t haplotypes

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7 January 2021

Comparison of the cloned H-2Kbm1 variant gene with the H-2Kb gene shows a cluster of seven nucleotide differences

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7 January 2021

Inversion in the H–2 complex of t-haplotypes in mice

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7 January 2021

Genetic structure and origin of t haplotypes of mice, analyzed with H-2 cDNA probes

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7 January 2021

A new wild-derived H-2 haplotype enhancing K-IA recombination.

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7 January 2021

Genetic organization of the mouse t complex

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7 January 2021

Genomic analysis of the H-2 complex region associated with mouse t haplotypes

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7 January 2021

Persistence of a lethal t haplotype in a laboratory stock of outbred mice

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7 January 2021

A pseudogene homologous to mouse transplantation antigens: transplantation antigens are encoded by eight exons that correlate with protein domains

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7 January 2021

Clusters of genes encoding mouse transplantation antigens

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7 January 2021

Organization and evolution of the class I gene family in the major histocompatibility complex of the C57BL/10 mouse

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7 January 2021

Biochemical studies of H-2K antigens from a group of related mutants. I. Identification of a shared mutation in B6-H-2bm5 and B6-H-2bm16

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7 January 2021

Biochemical studies of H-2K antigens from a group of related mutants. II. Identification of a shared mutation in B6-H-2bm6, B6.C-H-2bm7, and B6.C-H-2bm9

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7 January 2021

Genetic mapping in the major histocompatibility complex by restriction enzyme site polymorphisms: most mouse class I genes map to the Tla complex

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7 January 2021

10.1016/S0769-2625(85)80039-8

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DOI

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