25-Hydroxy vitamin D3 modulates dendritic cell phenotype and function in Crohn’s disease (Q63727323)

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22 February 2020

title

25-Hydroxy vitamin D3 modulates dendritic cell phenotype and function in Crohn's disease (English)

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22 February 2020

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22 February 2020

Christian L Hvas

4

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22 February 2020

Jørgen Agnholt

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22 February 2020

Jens F. Dahlerup

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22 February 2020

author name string

Ralf Agger

6

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22 February 2020

Lars E Bartels

1

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22 February 2020

Søren P Jørgensen

2

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22 February 2020

publication date

23 January 2013

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22 February 2020

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22 February 2020

volume

21

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22 February 2020

issue

2

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22 February 2020

page(s)

177-186

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https://scigraph.springernature.com/pub.10.1007/s10787-012-0168-y

22 February 2020

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cites work

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1,25-dihydroxyvitamin D3 and dexamethasone increase interleukin-10 production in CD4+ T cells from patients with Crohn's disease

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Injury enhances TLR2 function and antimicrobial peptide expression through a vitamin D-dependent mechanism

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Patients with active inflammatory bowel disease lack immature peripheral blood plasmacytoid and myeloid dendritic cells

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Measurement of vitamin D levels in inflammatory bowel disease patients reveals a subset of Crohn's disease patients with elevated 1,25-dihydroxyvitamin D and low bone mineral density

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Human dendritic cell antigen presentation and chemotaxis are inhibited by intrinsic 25-hydroxy vitamin D activation

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1 Alpha,25-dihydroxyvitamin D3 inhibits differentiation, maturation, activation, and survival of dendritic cells leading to impaired alloreactive T cell activation

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Increased expression of DC-SIGN+IL-12+IL-18+ and CD83+IL-12-IL-18- dendritic cell populations in the colonic mucosa of patients with Crohn's disease.

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