Mechanism of Interaction of Niflumic Acid with Heterologously Expressed Kidney CLC-K Chloride Channels (Q60330900)

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2 January 2020

title

Mechanism of interaction of niflumic acid with heterologously expressed kidney CLC-K chloride channels (English)

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1

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Antonella Liantonio

2

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Michael Pusch

5

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author name string

Elena Babini

3

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Diana Conte Camerino

4

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publication date

1 April 2007

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Journal of Membrane Biology

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published in

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volume

216

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issue

2-3

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page(s)

73-82

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https://scigraph.springernature.com/pub.10.1007/s00232-007-9034-z

2 January 2020

exact match

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Voltage-dependent block of the cystic fibrosis transmembrane conductance regulator Cl- channel by two closely related arylaminobenzoates

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12 December 2020

Mutations in the chloride channel gene, CLCNKB, cause Bartter's syndrome type III

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Two isoforms of a chloride channel predominantly expressed in thick ascending limb of Henle's loop and collecting ducts of rat kidney

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Molecular determinants of differential pore blocking of kidney CLC-K chloride channels

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Two highly homologous members of the ClC chloride channel family in both rat and human kidney

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Investigations of pharmacologic properties of the renal CLC-K1 chloride channel co-expressed with barttin by the use of 2-(p-Chlorophenoxy)propionic acid derivatives and other structurally unrelated chloride channels blockers

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12 December 2020

Localization and induction by dehydration of ClC-K chloride channels in the rat kidney.

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12 December 2020

Niflumic and flufenamic acids are potent reversible blockers of Ca2(+)-activated Cl- channels in Xenopus oocytes

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12 December 2020

Flufenamic acid, mefenamic acid and niflumic acid inhibit single nonselective cation channels in the rat exocrine pancreas

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12 December 2020

Function of chloride channels in the kidney

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12 December 2020

Pharmacological characterization of chloride channels belonging to the ClC family by the use of chiral clofibric acid derivatives.

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12 December 2020

Overt nephrogenic diabetes insipidus in mice lacking the CLC-K1 chloride channel.

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Anion permeation in Ca(2+)-activated Cl(-) channels

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Unique structure and function of chloride transporting CLC proteins

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ClC-6 and ClC-7 are two novel broadly expressed members of the CLC chloride channel family

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Activation and inhibition of kidney CLC-K chloride channels by fenamates

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Barttin is a Cl- channel beta-subunit crucial for renal Cl- reabsorption and inner ear K+ secretion

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Biochemically based design of cyclooxygenase-2 (COX-2) inhibitors: facile conversion of nonsteroidal antiinflammatory drugs to potent and highly selective COX-2 inhibitors

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Inhibition of hKv2.1, a major human neuronal voltage-gated K+ channel, by meclofenamic acid

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Mutation of BSND causes Bartter syndrome with sensorineural deafness and kidney failure

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10.1007/S00232-007-9034-Z

Identifiers

DOI

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