Effect of changes in action potential spike configuration, junctional sarcoplasmic reticulum micro-architecture and altered t-tubule structure in human heart failure (Q57923269)

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article by M. B. Cannell et al published 4 August 2006 in Journal of Muscle Research & Cell Motility

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English	Effect of changes in action potential spike configuration, junctional sarcoplasmic reticulum micro-architecture and altered t-tubule structure in human heart failure	article by M. B. Cannell et al published 4 August 2006 in Journal of Muscle Research & Cell Motility

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scholarly article

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Effect of changes in action potential spike configuration, junctional sarcoplasmic reticulum micro-architecture and altered t-tubule structure in human heart failure (English)

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author name string

M. B. Cannell

1

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D. J. Crossman

2

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C. Soeller

3

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publication date

4 August 2006

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Journal of Muscle Research & Cell Motility

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27

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297-306

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5-7

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https://scigraph.springernature.com/pub.10.1007/s10974-006-9089-y

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Global gene expression profiling of end-stage dilated cardiomyopathy using a human cardiovascular-based cDNA microarray

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Alterations of K+ currents in isolated human ventricular myocytes from patients with terminal heart failure

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Fluspirilene block of N-type calcium current in NGF-differentiated PC12 cells

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Molecular basis of transient outward potassium current downregulation in human heart failure: a decrease in Kv4.3 mRNA correlates with a reduction in current density

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Dihydropyridine receptors are primarily functional L-type calcium channels in rabbit ventricular myocytes

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Distribution, surface density, and membrane area of diadic junctional contacts between plasma membrane and terminal cisterns in mammalian ventricle

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Local Ca2+ transients (Ca2+ sparks) originate at transverse tubules in rat heart cells

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Patients with end-stage congestive heart failure treated with beta-adrenergic receptor antagonists have improved ventricular myocyte calcium regulatory protein abundance

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Impairment of the myocardial ultrastructure and changes of the cytoskeleton in dilated cardiomyopathy

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Time and calcium dependence of activation and inactivation of calcium-induced release of calcium from the sarcoplasmic reticulum of a skinned canine cardiac Purkinje cell

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A computational model of the human left-ventricular epicardial myocyte

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Intracellular calcium handling in isolated ventricular myocytes from patients with terminal heart failure

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Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced heart failure.

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The control of calcium release in heart muscle

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Heart failure in pressure overload hypertrophy. The relative roles of ventricular remodeling and myocardial dysfunction

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Numerical simulation of local calcium movements during L-type calcium channel gating in the cardiac diad

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EC-coupling in normal and failing hearts

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Potassium channel down-regulation in heart failure.

1 reference

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A simple numerical model of calcium spark formation and detection in cardiac myocytes

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https://pubmed.ncbi.nlm.nih.gov/16897575

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Mechanism of Ca(2+)-sensitive inactivation of L-type Ca2+ channels.

1 reference

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Spatial non-uniformities in [Ca2+]i during excitation-contraction coupling in cardiac myocytes

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Phospholamban: a key determinant of cardiac function and dysfunction.

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Local control model of excitation-contraction coupling in skeletal muscle

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Cardiac excitation-contraction coupling

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Termination of cardiac Ca(2+) sparks: an investigative mathematical model of calcium-induced calcium release

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https://pubmed.ncbi.nlm.nih.gov/16897575

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Local calcium transients triggered by single L-type calcium channel currents in cardiac cells

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A mathematical analysis of the generation and termination of calcium sparks

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Defective excitation-contraction coupling in experimental cardiac hypertrophy and heart failure

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Electrical heterogeneity within the ventricular wall

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Quantitative electron microscopic description of heart muscle cells. Application to normal, hypertrophied and thyroxin-stimulated hearts.

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Propagation of excitation-contraction coupling into ventricular myocytes

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Cellular mechanisms of altered contractility in the hypertrophied heart: big hearts, big sparks

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Cellular basis of abnormal calcium transients of failing human ventricular myocytes

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Sparks of interest in cardiac excitation–contraction coupling

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Numerical analysis of ryanodine receptor activation by L-type channel activity in the cardiac muscle diad

1 reference

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1 reference

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Reduced synchrony of Ca2+ release with loss of T-tubules-a comparison to Ca2+ release in human failing cardiomyocytes

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https://pubmed.ncbi.nlm.nih.gov/16897575

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Calcium sparks and [Ca2+]i waves in cardiac myocytes

1 reference

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Ultrastructural features of degenerated cardiac muscle cells in patients with cardiac hypertrophy

1 reference

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Calcium sparks: elementary events underlying excitation-contraction coupling in heart muscle

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

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The role of action potential prolongation and altered intracellular calcium handling in the pathogenesis of heart failure.

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

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based on heuristic

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Theory of excitation-contraction coupling in cardiac muscle

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

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Reduced contraction strength with increased intracellular [Ca2+] in left ventricular trabeculae from failing rat hearts

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Depletion of T-tubules and specific subcellular changes in sarcolemmal proteins in tachycardia-induced heart failure.

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The cytoskeleton and related proteins in the human failing heart

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Functional properties of failing human ventricular myocytes

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Orphaned ryanodine receptors in the failing heart.

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

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Dyssynchronous Ca(2+) sparks in myocytes from infarcted hearts

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https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

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Modulation of Ca(2+) release in cardiac myocytes by changes in repolarization rate: role of phase-1 action potential repolarization in excitation-contraction coupling

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

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The gene expression fingerprint of human heart failure

1 reference

https://pubmed.ncbi.nlm.nih.gov/16897575

12 December 2020

based on heuristic

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Properties of Ca2+ sparks evoked by action potentials in mouse ventricular myocytes

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10.1007/S10974-006-9089-Y

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