Uncoupling Protein 2 Promoter Polymorphism −866G/A, Central Adiposity, and Metabolic Syndrome in Asians* (Q57337116)

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object named as

Lu Qi

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author name string

Haiqing Shen

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E. Shyong Tai

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Suok Kai Chew

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Chee Eng Tan

Uncoupling protein-2: a novel gene linked to obesity and hyperinsulinemia

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publication date

April 2006

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published in

Obesity

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volume

14

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issue

4

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page(s)

656-661

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cites work

1 reference

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A significant portion of mitochondrial proton leak in intact thymocytes depends on expression of UCP2.

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Uncoupling protein 2 and islet function

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Uncoupling protein-2 gene: reduced mRNA expression in intraperitoneal adipose tissue of obese humans

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A common polymorphism in the promoter of UCP2 is associated with decreased risk of obesity in middle-aged humans

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A common polymorphism of uncoupling protein 2 gene is associated with hypertension.

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A polymorphism in the promoter of UCP2 gene modulates lipid levels in patients with type 2 diabetes

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The -866A/A genotype in the promoter of the human uncoupling protein 2 gene is associated with insulin resistance and increased risk of type 2 diabetes.

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A functional polymorphism in the promoter of UCP2 enhances obesity risk but reduces type 2 diabetes risk in obese middle-aged humans.

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Uncoupling protein 2 promoter polymorphism -866G/A affects its expression in beta-cells and modulates clinical profiles of Japanese type 2 diabetic patients

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Uncoupling protein-2 polymorphisms in type 2 diabetes, obesity, and insulin secretion

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Mutational analysis of the UCP2 core promoter and relationships of variants with obesity

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Sexual dimorphism in age-related changes in UCP2 and leptin gene expression in subcutaneous adipose tissue in humans

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Opposite actions of testosterone and progesterone on UCP1 mRNA expression in cultured brown adipocytes.

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The common -866G/A polymorphism in the promoter region of the UCP-2 gene is associated with reduced risk of type 2 diabetes in Caucasians from Italy

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The TaqIB and -629C>A polymorphisms at the cholesteryl ester transfer protein locus: associations with lipid levels in a multiethnic population. The 1998 Singapore National Health Survey

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Can we apply the National Cholesterol Education Program Adult Treatment Panel definition of the metabolic syndrome to Asians?

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Physiological role of UCP3 may be export of fatty acids from mitochondria when fatty acid oxidation predominates: an hypothesis

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