CTLA4 is associated with susceptibility to multiple sclerosis (Q56904087)

1

Orhun H Kantarci

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Elizabeth J Atkinson

4

Elizabeth J Atkinson

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Mariza de Andrade

8

Mariza de Andrade

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Brian G Weinshenker

10

Brian G Weinshenker

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Cynthia T McMurray

7

Cynthia T McMurray

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author name string

David D Hebrink

2

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Sara J Achenbach

3

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Alicja Waliszewska

5

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Guy Buckle

Journal of Neuroimmunology

6

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language of work or name

English

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publication date

January 2003

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published in

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volume

134

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issue

1-2

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page(s)

133-41

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133-141

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cites work

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Long-term inhibition of murine experimental autoimmune encephalomyelitis using CTLA-4-Fc supports a key role for CD28 costimulation.

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Induction of cytotoxic T lymphocyte antigen 4 (CTLA-4) restricts clonal expansion of helper T cells

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No evidence to support CTLA-4 as a susceptibility gene in MS families: the Canadian Collaborative Study

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A full genome search in multiple sclerosis

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CTLA-4 gene polymorphism may modulate disease in Japanese multiple sclerosis patients.

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Human T-cell clonal anergy is induced by antigen presentation in the absence of B7 costimulation

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Association of two variants in IL-1beta and IL-1 receptor antagonist genes with multiple sclerosis

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CTLA-4 engagement inhibits IL-2 accumulation and cell cycle progression upon activation of resting T cells

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Genomewide scan of multiple sclerosis in Finnish multiplex families

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The CTLA-4 gene is associated with multiple sclerosis.

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The CTLA-4 gene region of chromosome 2q33 is linked to, and associated with, type 1 diabetes. Belgian Diabetes Registry

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CTLA4 in multiple sclerosis. Lack of genetic association in a European Caucasian population but evidence of interaction with HLA-DR2 among Shanghai Chinese

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