Characterization of a familial case with primary erythromelalgia from Taiwan. (Q55043177)

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25 December 2019

title

Characterization of a familial case with primary erythromelalgia from Taiwan (English)

1 reference

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25 December 2019

6

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25 December 2019

Ming-Jen Lee

1

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25 December 2019

Sung-Tsang Hsieh

3

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25 December 2019

author name string

Hsin-Su Yu

2

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25 December 2019

Dennis A Stephenson

4

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25 December 2019

Chien-Jung Lu

5

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publication date

8 February 2007

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25 December 2019

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25 December 2019

volume

254

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issue

2

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25 December 2019

page(s)

210-214

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https://scigraph.springernature.com/pub.10.1007/s00415-006-0328-3

25 December 2019

exact match

0 references

cites work

Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy.

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12 December 2020

The primary erythermalgia-susceptibility gene is located on chromosome 2q31-32.

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Erythromelalgia: vasculopathy, neuropathy, or both? A prospective study of vascular and neurophysiologic studies in erythromelalgia

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Lidocaine patch for pain of erythromelalgia.

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12 December 2020

SCN9A mutations define primary erythermalgia as a neuropathic disorder of voltage gated sodium channels

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12 December 2020

Impaired neurogenic control of skin perfusion in erythromelalgia

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12 December 2020

Lidocaine and mexiletine therapy for erythromelalgia.

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12 December 2020

Erythromelalgia: a hereditary pain syndrome enters the molecular era.

1 reference

12 December 2020

Sporadic onset of erythermalgia: a gain-of-function mutation in Nav1.7.

1 reference

12 December 2020

Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

1 reference

12 December 2020

Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

1 reference

12 December 2020

Autonomic Innervation of the Skin in Primary Erythermalgia

1 reference

12 December 2020