Resolution of neutrophilic inflammation by H2O2 in antigen-induced arthritis. (Q54590358)

1 June 2018

title

Resolution of neutrophilic inflammation by H2O2 in antigen-induced arthritis. (English)

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1 June 2018

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5

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Tarcilia Aparecida Silva

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Vivian V Costa

object named as

Vivian V Costa

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Vanessa Pinho

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Vanessa Pinho

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Mauro M Teixeira

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object named as

Mauro M Teixeira

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1 June 2018

author name string

Fernando Lopes

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Fernanda M Coelho

2

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Érica L M Vieira

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Leda Q Vieira

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publication date

1 September 2011

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1 June 2018

published in

Arthritis and Rheumatism

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1 June 2018

volume

63

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1 June 2018

page(s)

2651-2660

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issue

9

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Evolving concepts of rheumatoid arthritis

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The costs of arthritis

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Neutrophil-derived cytokines: potential therapeutic targets in inflammation.

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Immunologic mechanisms in the pathogenesis of rheumatoid arthritis

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The chemokine receptors CXCR1/CXCR2 modulate antigen-induced arthritis by regulating adhesion of neutrophils to the synovial microvasculature

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Essential role of neutrophils in the initiation and progression of a murine model of rheumatoid arthritis.

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Glutathione reductase plays an anti-apoptotic role against oxidative stress in human hepatoma cells

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Heat shock transcription factor-1 inhibits H2O2-induced apoptosis via down-regulation of reactive oxygen species in cardiac myocytes.

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Production and in vivo effects of chemokines CXCL1-3/KC and CCL2/JE in a model of inflammatory angiogenesis in mice.

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Phosphoinositide-3 kinases critically regulate the recruitment and survival of eosinophils in vivo: importance for the resolution of allergic inflammation

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Neuronal nitric oxide synthase-derived hydrogen peroxide is a major endothelium-dependent relaxing factor.

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Pathways involved in testicular germ cell apoptosis induced by H2O2 in vitro

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Deguelin, an Akt inhibitor, suppresses IkappaBalpha kinase activation leading to suppression of NF-kappaB-regulated gene expression, potentiation of apoptosis, and inhibition of cellular invasion.

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NF-kappaB plays a major role during the systemic and local acute inflammatory response following intestinal reperfusion injury

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Identifiers

DOI

10.1002/ART.30448

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1 June 2018

PubMed ID

21567381

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1 June 2018