Enhanced recovery from ischemia-reperfusion injury in PI3Kα dominant negative hearts: investigating the role of alternate PI3K isoforms, increased glucose oxidation and MAPK signaling. (Q54472460)

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English	Enhanced recovery from ischemia-reperfusion injury in PI3Kα dominant negative hearts: investigating the role of alternate PI3K isoforms, increased glucose oxidation and MAPK signaling.	scientific article

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17 February 2020

Enhanced recovery from ischemia-reperfusion injury in PI3Kα dominant negative hearts: investigating the role of alternate PI3K isoforms, increased glucose oxidation and MAPK signaling (English)

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17 February 2020

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reperfusion injury

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based on heuristic

inferred from title

Petra C Kienesberger

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17 February 2020

Pavel Zhabyeyev

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17 February 2020

author name string

Brent A McLean

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17 February 2020

Wang Wang

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Grant Masson

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Jason R B Dyck

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Gavin Y Oudit

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8 November 2012

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17 February 2020

Journal of Molecular and Cellular Cardiology

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17 February 2020

54

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9-18

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17 February 2020

Myocardial reperfusion injury.

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Left ventricular remodeling after myocardial infarction: pathophysiology and therapy

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The reperfusion injury salvage kinase pathway: a common target for both ischemic preconditioning and postconditioning.

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Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase--dependent pathway is cardioprotective

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Regulation of myocardial contractility and cell size by distinct PI3K-PTEN signaling pathways

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Phosphatidylinositol 3-kinase gamma is a critical mediator of myocardial ischemic and adenosine-mediated preconditioning

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PI3Kgamma protects from myocardial ischemia and reperfusion injury through a kinase-independent pathway

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7 January 2021

Reperfusion-activated Akt kinase prevents apoptosis in transgenic mouse hearts overexpressing insulin-like growth factor-1

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New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway

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7 January 2021

Phosphoinositide 3-kinase(p110α) plays a critical role for the induction of physiological, but not pathological, cardiac hypertrophy

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7 January 2021

PI3K(p110 alpha) protects against myocardial infarction-induced heart failure: identification of PI3K-regulated miRNA and mRNA.

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Cardiac regulation by phosphoinositide 3-kinases and PTEN

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7 January 2021

Protective effects of exercise and phosphoinositide 3-kinase(p110alpha) signaling in dilated and hypertrophic cardiomyopathy

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Wortmannin and its structural analogue demethoxyviridin inhibit stimulated phospholipase A2 activity in Swiss 3T3 cells. Wortmannin is not a specific inhibitor of phosphatidylinositol 3-kinase.

1 reference

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7 January 2021

Wortmannin, a widely used phosphoinositide 3-kinase inhibitor, also potently inhibits mammalian polo-like kinase.

1 reference

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7 January 2021

Loss of PI3Kγ enhances cAMP-dependent MMP remodeling of the myocardial N-cadherin adhesion complexes and extracellular matrix in response to early biomechanical stress

1 reference

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7 January 2021

Loss of PTEN attenuates the development of pathological hypertrophy and heart failure in response to biomechanical stress

1 reference

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7 January 2021

Angiotensin-converting enzyme 2 suppresses pathological hypertrophy, myocardial fibrosis, and cardiac dysfunction.

1 reference

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7 January 2021

Fatty acid translocase/CD36 deficiency does not energetically or functionally compromise hearts before or after ischemia

1 reference

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7 January 2021

Absence of malonyl coenzyme A decarboxylase in mice increases cardiac glucose oxidation and protects the heart from ischemic injury

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

Role of glycogen synthase kinase-3beta in cardioprotection

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

The conserved phosphoinositide 3-kinase pathway determines heart size in mice

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

PI3Ks maintain the structural integrity of T-tubules in cardiac myocytes

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

The Akt1 isoform is an essential mediator of ischaemic preconditioning

1 reference

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7 January 2021

The p110beta isoform of phosphoinositide 3-kinase signals downstream of G protein-coupled receptors and is functionally redundant with p110gamma.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

Synergistic activation of a family of phosphoinositide 3-kinase via G-protein coupled and tyrosine kinase-related receptors

1 reference

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7 January 2021

Hypoglycaemia, liver necrosis and perinatal death in mice lacking all isoforms of phosphoinositide 3-kinase p85 alpha

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

High levels of fatty acids delay the recovery of intracellular pH and cardiac efficiency in post-ischemic hearts by inhibiting glucose oxidation

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

Myocardial fatty acid metabolism in health and disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

Stimulation of glucose oxidation protects against acute myocardial infarction and reperfusion injury

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

Effects of chronic Akt activation on glucose uptake in the heart

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

A conserved role for phosphatidylinositol 3-kinase but not Akt signaling in mitochondrial adaptations that accompany physiological cardiac hypertrophy.

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2012.10.015

7 January 2021

The glucose fatty-acid cycle. Its role in insulin sensitivity and the metabolic disturbances of diabetes mellitus

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AMPK alterations in cardiac physiology and pathology: enemy or ally?

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7 January 2021

AMPK activation, a preventive therapeutic target in the transition from cardiac injury to heart failure

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7 January 2021

High rates of fatty acid oxidation during reperfusion of ischemic hearts are associated with a decrease in malonyl-CoA levels due to an increase in 5'-AMP-activated protein kinase inhibition of acetyl-CoA carboxylase

1 reference

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7 January 2021

Phosphorylation control of cardiac acetyl-CoA carboxylase by cAMP-dependent protein kinase and 5'-AMP activated protein kinase

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AMPK-Mediated AS160 Phosphorylation in Skeletal Muscle Is Dependent on AMPK Catalytic and Regulatory Subunits

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MEK1-ERK2 signaling pathway protects myocardium from ischemic injury in vivo.

1 reference

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Involvement of extracellular signal-regulated kinases 1/2 in cardiac hypertrophy and cell death

1 reference

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Activation of p38 mitogen-activated protein kinase abolishes insulin-mediated myocardial protection against ischemia-reperfusion injury

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7 January 2021

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AKT inhibition relieves feedback suppression of receptor tyrosine kinase expression and activity

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10.1016/J.YJMCC.2012.10.015

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17 February 2020

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Europe PubMed Central

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17 February 2020

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