Oxidative imbalance and cathepsin D changes as peripheral blood biomarkers of Alzheimer disease: a pilot study. (Q53269766)

12 May 2018

title

Oxidative imbalance and cathepsin D changes as peripheral blood biomarkers of Alzheimer disease: a pilot study. (English)

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12 May 2018

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Walter Malorni

object named as

Walter Malorni

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author name string

Elisabetta Straface

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Paola Matarrese

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Lucrezia Gambardella

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Antonio Sgadari

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Maria Caterina Silveri

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language of work or name

English

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publication date

19 April 2005

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12 May 2018

number of pages

8

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based on heuristic

inferred from page(s)

published in

FEBS Letters

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volume

579

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issue

13

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page(s)

2759-2766

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Up-regulation of the lysosomal system in experimental models of neuronal injury: implications for Alzheimer's disease.

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Cathepsin D triggers Bax activation, resulting in selective apoptosis-inducing factor (AIF) relocation in T lymphocytes entering the early commitment phase to apoptosis

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Cytochrome c oxidase is decreased in Alzheimer's disease platelets

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Lysosomal hydrolases of different classes are abnormally distributed in brains of patients with Alzheimer disease

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Identification of oxidized plasma proteins in Alzheimer's disease

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Platelets as a peripheral district where to study pathogenetic mechanisms of alzheimer disease: the case of amyloid precursor protein.

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The effect of increased concentrations of homocysteine on the concentration of (E)-4-hydroxy-2-nonenal in the plasma and cerebrospinal fluid of patients with Alzheimer's disease.

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Amyloid β/A4 Precursor Protein (APP) Processing in Lysosomesa

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Cystatin C colocalizes with amyloid-beta and coimmunoprecipitates with amyloid-beta precursor protein in sporadic inclusion-body myositis muscles.

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21 January 2018

Identifiers

DOI

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12 May 2018

PubMed ID

15907478

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12 May 2018