Downregulation of protein phosphatase 2A carboxyl methylation and methyltransferase may contribute to Alzheimer disease pathogenesis. (Q53265449)

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4 January 2020

title

Downregulation of protein phosphatase 2A carboxyl methylation and methyltransferase may contribute to Alzheimer disease pathogenesis (English)

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4 January 2020

6

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7

Charles L White 3rd

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4 January 2020

author name string

Estelle Sontag

1

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4 January 2020

Christa Hladik

2

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4 January 2020

Lisa Montgomery

3

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4 January 2020

Ampa Luangpirom

4

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4 January 2020

Ingrid Mudrak

5

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publication date

1 October 2004

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Journal of Neuropathology & Experimental Neurology

4 January 2020

published in

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volume

63

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issue

10

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4 January 2020

page(s)

1080-1091

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Protein phosphatase 2A: the Trojan Horse of cellular signaling

4 January 2020

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Protein phosphatase 2A subunit assembly: the catalytic subunit carboxy terminus is important for binding cellular B subunit but not polyomavirus middle tumor antigen

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Carboxyl methylation regulates phosphoprotein phosphatase 2A by controlling the association of regulatory B subunits

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Carboxyl methylation of the phosphoprotein phosphatase 2A catalytic subunit promotes its functional association with regulatory subunits in vivo

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Methylation of the protein phosphatase 2A catalytic subunit is essential for association of Balpha regulatory subunit but not SG2NA, striatin, or polyomavirus middle tumor antigen

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Carboxymethylation of the PP2A catalytic subunit in Saccharomyces cerevisiae is required for efficient interaction with the B-type subunits Cdc55p and Rts1p

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A specific protein carboxyl methylesterase that demethylates phosphoprotein phosphatase 2A in bovine brain

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A protein phosphatase methylesterase (PME-1) is one of several novel proteins stably associating with two inactive mutants of protein phosphatase 2A

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Structure of protein phosphatase methyltransferase 1 (PPM1), a leucine carboxyl methyltransferase involved in the regulation of protein phosphatase 2A activity

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Inhibition of protein phosphatase 2A overrides tau protein kinase I/glycogen synthase kinase 3 beta and cyclin-dependent kinase 5 inhibition and results in tau hyperphosphorylation in the hippocampus of starved mouse

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Impairment of phosphatase 2A contributes to the prolonged MAP kinase phosphorylation in Alzheimer's disease fibroblasts

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Differential methylation and altered conformation of cytoplasmic and nuclear forms of protein phosphatase 2A during cell cycle progression

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Optimization of techniques for the maximal detection and quantification of Alzheimer's-related neuropathology with digital imaging

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