Cytoprotective regulation of the mitochondrial permeability transition pore is impaired in type 2 diabetic Goto-Kakizaki rat hearts. (Q53139901)

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10 March 2020

title

Cytoprotective regulation of the mitochondrial permeability transition pore is impaired in type 2 diabetic Goto-Kakizaki rat hearts (English)

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5

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author name string

Takahito Itoh

1

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Hidemichi Kouzu

2

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Takayuki Miki

3

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Masaya Tanno

4

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Tatsuya Sato

6

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10 March 2020

Daisuke Sunaga

7

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Hiromichi Murase

8

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Tetsuji Miura

9

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publication date

12 October 2012

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Journal of Molecular and Cellular Cardiology

10 March 2020

published in

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volume

53

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issue

6

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page(s)

870-879

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Diabetes in heart failure: prevalence and impact on outcome in the population

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Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transition

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Modulation of the mitochondrial permeability transition pore complex in GSK-3beta-mediated myocardial protection

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Hypertensive hypertrophied myocardium is vulnerable to infarction and refractory to erythropoietin-induced protection.

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The mitochondrial phosphate carrier interacts with cyclophilin D and may play a key role in the permeability transition

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Identifiers

DOI

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10 March 2020

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