An EGFR/Ebi/Sno pathway promotes delta expression by inactivating Su(H)/SMRTER repression during inductive notch signaling. (Q52599088)

4 May 2018

title

An EGFR/Ebi/Sno pathway promotes delta expression by inactivating Su(H)/SMRTER repression during inductive notch signaling. (English)

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4 May 2018

Strawberry notch Dmel_CG44436

main subject

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GOA release 2020-03-11

Ebi Dmel_CG4063

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GOA release 2020-03-11

author name string

Leo Tsuda

1

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4 May 2018

Raghavendra Nagaraj

2

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4 May 2018

S Lawrence Zipursky

3

1 reference

4 May 2018

Utpal Banerjee

4

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4 May 2018

language of work or name

English

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publication date

1 September 2002

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4 May 2018

published in

Cell

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4 May 2018

volume

110

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4 May 2018

page(s)

625-637

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4 May 2018

issue

5

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4 May 2018

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The EGF receptor defines domains of cell cycle progression and survival to regulate cell number in the developing Drosophila eye.

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A notch-independent activity of suppressor of hairless is required for normal mechanoreceptor physiology

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A role for Ebi in neuronal cell cycle control

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Notch is required for successive cell decisions in the developing Drosophila retina.

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Reciprocal regulatory interactions between the Notch and Ras signaling pathways in the Drosophila embryonic mesoderm.

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Control of EGF receptor signalling: lessons from fruitflies.

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ebi regulates epidermal growth factor receptor signaling pathways in Drosophila

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Combinatorial signaling in the specification of unique cell fates

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The spitz gene is required for photoreceptor determination in the Drosophila eye where it interacts with the EGF receptor

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In situ activation pattern of Drosophila EGF receptor pathway during development.

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The SMRT corepressor is regulated by a MEK-1 kinase pathway: inhibition of corepressor function is associated with SMRT phosphorylation and nuclear export

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A histone deacetylase corepressor complex regulates the Notch signal transduction pathway

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Ligand-induced cleavage and regulation of nuclear entry of Notch in Drosophila melanogaster embryos

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Integration of positional signals and regulation of wing formation and identity by Drosophila vestigial gene.

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EGF receptor and Notch signaling act upstream of Eyeless/Pax6 to control eye specification.

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The neurogenic suppressor of hairless DNA-binding protein mediates the transcriptional activation of the enhancer of split complex genes triggered by Notch signaling.

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Indirect evidence for Delta-dependent intracellular processing of notch in Drosophila embryos

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Several levels of EGF receptor signaling during photoreceptor specification in wild-type, Ellipse, and null mutant Drosophila

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Proneural enhancement by Notch overcomes Suppressor-of-Hairless repressor function in the developing Drosophila eye.

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Both corepressor proteins SMRT and N-CoR exist in large protein complexes containing HDAC3

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BCR/ABL inhibition by an escort/phosphatase fusion protein

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Siah-1, SIP, and Ebi collaborate in a novel pathway for beta-catenin degradation linked to p53 responses

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Transcriptional repression by suppressor of hairless involves the binding of a hairless-dCtBP complex in Drosophila.

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The activities of two Ets-related transcription factors required for Drosophila eye development are modulated by the Ras/MAPK pathway

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Relationships between complex Delta expression and the specification of retinal cell fates during Drosophila eye development.

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Notch-1 signalling requires ligand-induced proteolytic release of intracellular domain

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Sequential signalling during Caenorhabditis elegans vulval induction

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Nuclear access and action of notch in vivo

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spitz, a Drosophila homolog of transforming growth factor-alpha, is required in the founding photoreceptor cells of the compound eye facets.

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Delta/Notch and Boss/Sevenless signals act combinatorially to specify the Drosophila R7 photoreceptor

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SMRTER, a Drosophila nuclear receptor coregulator, reveals that EcR-mediated repression is critical for development.

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Reciprocal changes in expression of the receptor lin-12 and its ligand lag-2 prior to commitment in a C. elegans cell fate decision.

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Identifiers

DOI

1 reference

4 May 2018

PubMed ID

12230979

1 reference

4 May 2018