Chronic melatonin treatment reduces the age-dependent inflammatory process in senescence-accelerated mice. (Q51762701)

11 April 2018

title

Chronic melatonin treatment reduces the age-dependent inflammatory process in senescence-accelerated mice. (English)

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Darío Acuña-Castroviejo

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Luis C. López

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object named as

Luis C López

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author name string

María I Rodríguez

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Ana López

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José A García

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Francisco Ortiz

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publication date

1 April 2007

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Journal of Pineal Research

published in

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volume

42

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page(s)

272-279

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issue

3

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The effect of age on cyclooxygenase-2 gene expression: NF-kappaB activation and IkappaBalpha degradation

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A higher oxidative status accelerates senescence and aggravates age-dependent disorders in SAMP strains of mice

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Oxidative damage in the senescence-accelerated mouse.

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Changes in expressions of proinflammatory cytokines IL-1beta, TNF-alpha and IL-6 in the brain of senescence accelerated mouse (SAM) P8.

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Chronic melatonin treatment prevents age-dependent cardiac mitochondrial dysfunction in senescence-accelerated mice.

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Nitrate biosynthesis in man

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Melatonin, longevity and health in the aged: an assessment

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Inverse correlation between endogenous melatonin levels and oxidative damage in some tissues of SAM P8 mice.

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Circadian organization of the immune response: A role for melatonin

21 January 2018

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10.1111/J.1600-079X.2006.00416.X

21 January 2018

Identifiers

DOI

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11 April 2018

PubMed ID

17349026

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11 April 2018