Inducible expression of mutant human DISC1 in mice is associated with brain and behavioral abnormalities reminiscent of schizophrenia. (Q51738410)

11 April 2018

title

Inducible expression of mutant human DISC1 in mice is associated with brain and behavioral abnormalities reminiscent of schizophrenia. (English)

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11 April 2018

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Pletnikov MV

1

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11 April 2018

Ayhan Y

2

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11 April 2018

Nikolskaia O

3

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11 April 2018

Xu Y

4

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11 April 2018

Ovanesov MV

5

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11 April 2018

Huang H

6

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11 April 2018

Mori S

7

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11 April 2018

Moran TH

8

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11 April 2018

Ross CA

9

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11 April 2018

language of work or name

English

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publication date

11 September 2007

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11 April 2018

published in

Molecular Psychiatry

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11 April 2018

volume

13

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11 April 2018

page(s)

173-86, 115

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11 April 2018

issue

2

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11 April 2018

https://scigraph.springernature.com/pub.10.1038/sj.mp.4002079

exact match

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1 reference

https://api.crossref.org/works/10.1038%2FSJ.MP.4002079

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Genomic structure and localisation within a linkage hotspot of Disrupted In Schizophrenia 1, a gene disrupted by a translocation segregating with schizophrenia.

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Association within a family of a balanced autosomal translocation with major mental illness

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Disruption of two novel genes by a translocation co-segregating with schizophrenia

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Association of DISC1/TRAX haplotypes with schizophrenia, reduced prefrontal gray matter, and impaired short- and long-term memory

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Case-control association study of Disrupted-in-Schizophrenia-1 (DISC1) gene and schizophrenia in the Chinese population.

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Identification of polymorphisms within Disrupted in Schizophrenia 1 and Disrupted in Schizophrenia 2, and an investigation of their association with schizophrenia and bipolar affective disorder.

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Chromosome 1 loci in Finnish schizophrenia families

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A haplotype within the DISC1 gene is associated with visual memory functions in families with a high density of schizophrenia.

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Differential expression of disrupted-in-schizophrenia (DISC1) in bipolar disorder.

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Association between the TRAX/DISC locus and both bipolar disorder and schizophrenia in the Scottish population

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Association study of the DISC1/TRAX locus with schizophrenia in a Japanese population.

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Expression of Disrupted-In-Schizophrenia-1, a schizophrenia-associated gene, is prominent in the mouse hippocampus throughout brain development.

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Cloning and characterization of Disc1, the mouse ortholog of DISC1 (Disrupted-in-Schizophrenia 1).

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Disrupted-in-Schizophrenia-1 (DISC-1): mutant truncation prevents binding to NudE-like (NUDEL) and inhibits neurite outgrowth

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Yeast two-hybrid screens implicate DISC1 in brain development and function

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DISC1 (Disrupted-In-Schizophrenia 1) is a centrosome-associated protein that interacts with MAP1A, MIPT3, ATF4/5 and NUDEL: regulation and loss of interaction with mutation

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Disrupted in Schizophrenia 1 Interactome: evidence for the close connectivity of risk genes and a potential synaptic basis for schizophrenia

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Disrupted-In-Schizophrenia 1, a candidate gene for schizophrenia, participates in neurite outgrowth

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Disrupted in Schizophrenia 1 and Nudel form a neurodevelopmentally regulated protein complex: implications for schizophrenia and other major neurological disorders

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Subcellular targeting of DISC1 is dependent on a domain independent from the Nudel binding site.

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DISC1-NDEL1/NUDEL protein interaction, an essential component for neurite outgrowth, is modulated by genetic variations of DISC1

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DISC1 regulates neurotrophin-induced axon elongation via interaction with Grb2

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DISC1 regulates the transport of the NUDEL/LIS1/14-3-3epsilon complex through kinesin-1

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DISC1 and PDE4B are interacting genetic factors in schizophrenia that regulate cAMP signaling

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Evidence that many of the DISC1 isoforms in C57BL/6J mice are also expressed in 129S6/SvEv mice

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A schizophrenia-associated mutation of DISC1 perturbs cerebral cortex development

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Evolutionary constraints on the Disrupted in Schizophrenia locus

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Expression of DISC1 binding partners is reduced in schizophrenia and associated with DISC1 SNPs.

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Identifiers

DOI

10.1038/SJ.MP.4002079

1 reference

11 April 2018

PubMed ID

17848917

1 reference

11 April 2018