Possible involvement of nitric oxide in NMDA-induced glutamate release in the rat striatum: an in vivo microdialysis study. (Q51550049)

6 April 2018

title

Possible involvement of nitric oxide in NMDA-induced glutamate release in the rat striatum: an in vivo microdialysis study. (English)

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6 April 2018

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M B Bogdanov

1

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6 April 2018

R J Wurtman

series ordinal

2

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6 April 2018

language of work or name

English

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publication date

1 January 1997

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6 April 2018

number of pages

5

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inferred from page(s)

published in

Neuroscience Letters

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6 April 2018

volume

221

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6 April 2018

page(s)

197-201

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6 April 2018

issue

2-3

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6 April 2018

Nitric oxide mediates the sustained opening of NMDA receptor-gated ionic channels which follows transient excitotoxic exposure in hippocampal slices.

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Effects of systemic or oral ad libitum monosodium glutamate administration on striatal glutamate release, as measured using microdialysis in freely moving rats

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Nitric oxide mediates glutamate-linked enhancement of cGMP levels in the cerebellum

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NMDA-induced release of nitric oxide potentiates aspartate overflow from cerebellar slices.

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Endothelium-derived relaxing factor release on activation of NMDA receptors suggests role as intercellular messenger in the brain

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L-NAME modulates glutamate accumulation induced by K(+)-depolarization but not by forebrain ischaemia in the rat striatum.

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Modulation of In Vivo Striatal Transmitter Release by Nitric Oxide and Cyclic GMP

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Positive feedback of glutamate exocytosis by metabotropic presynaptic receptor stimulation

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Nitric oxide inhibition of the depolarization-evoked glutamate release from synaptosomes of rat cerebellum

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Calcium-dependent component of massive increase in extracellular potassium during cerebral ischemia as demonstrated by microdialysis in vivo

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Effect of nitric oxide production on the redox modulatory site of the NMDA receptor-channel complex

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N-methyl-D-aspartate-induced nitric oxide release: an in vivo microdialysis study

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Nitric oxide-induced blockade of NMDA receptors

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Nitric oxide stimulates Ca(2+)-independent synaptic vesicle release.

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Synaptically activated increases in Ca2+ concentration in hippocampal CA1 pyramidal cells are primarily due to voltage-gated Ca2+ channels

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Role of NO production in NMDA receptor-mediated neurotransmitter release in cerebral cortex

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A glutamate receptor regulates Ca2+ mobilization in hippocampal neurons

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Two pathways of cyclic GMP production through glutamate receptor-mediated nitric oxide synthesis

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Nitric oxide inhibits 3H-glutamate transport in synaptosomes.

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Nitric oxide and synaptic function

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N-Methyl-D-Aspartate Releases Excitatory Amino Acids in Rat Corpus Striatum In Vivo

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Identifiers

DOI

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6 April 2018

PubMed ID

9121698

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6 April 2018