Complement system becomes activated by the classical pathway in intracranial aneurysm walls. (Q50590763)

16 March 2018

title

Complement system becomes activated by the classical pathway in intracranial aneurysm walls. (English)

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16 March 2018

author

Seppo Meri

9

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16 March 2018

Mika Niemelä

object named as

Mika Niemelä

8

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Riikka Tulamo

object named as

Riikka Tulamo

1

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Juhana Frösen

2

object named as

Juhana Frösen

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16 March 2018

author name string

Sami Junnikkala

3

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16 March 2018

Anders Paetau

4

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16 March 2018

Marko Kangasniemi

5

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16 March 2018

Jose Peláez

6

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16 March 2018

Juha Hernesniemi

7

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16 March 2018

publication date

7 December 2009

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16 March 2018

published in

Laboratory Investigation

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16 March 2018

volume

90

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16 March 2018

issue

2

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16 March 2018

page(s)

168-179

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16 March 2018

https://scigraph.springernature.com/pub.10.1038/labinvest.2009.133

exact match

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cites work

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Discrimination between activators and nonactivators of the alternative pathway of complement: regulation via a sialic acid/polyanion binding site on factor H

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Proteases of the complement system

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Complement activation and inhibition: a delicate balance.

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Identification of the target self-antigens in reperfusion injury

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Topological determinants and consequences of adventitial responses to arterial wall injury.

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Colocalisation of intraplaque C reactive protein, complement, oxidised low density lipoprotein, and macrophages in stable and unstable angina and acute myocardial infarction

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Significance of glomerular deposition of C3c and C3d in IgA nephropathy

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Role of apoptosis in intracranial aneurysm rupture

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Natural history of unruptured intracranial aneurysms: probability of and risk factors for aneurysm rupture

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Mechanism of Growth and Rupture in Cerebral Berry Aneurysms

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Remodeling of Saccular Cerebral Artery Aneurysm Wall Is Associated With Rupture

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Inflammation and intracranial aneurysms

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Complement activation associates with saccular cerebral artery aneurysm wall degeneration and rupture.

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Growth Factor Receptor Expression and Remodeling of Saccular Cerebral Artery Aneurysm Walls: Implications for Biological Therapy Preventing Rupture

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NF-kappaB is a key mediator of cerebral aneurysm formation

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Modified low density lipoproteins differentially bind and activate the C1 complex of complement

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Antisera and monoclonal antibodies specific for epitopes generated during oxidative modification of low density lipoprotein.

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Common binding region of complement factors B, H and CR1 on C3b revealed by monoclonal anti-C3d

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Conservation of the low density lipoprotein receptor-binding domain of apoprotein B. Demonstration by a new monoclonal antibody, MB47

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Complement activation and subclassification of tissue immunoglobulin G in the abdominal aortic aneurysm

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https://api.crossref.org/works/10.1038%2FLABINVEST.2009.133

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Complement regulation in human atherosclerotic coronary lesions

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Endothelial injury and inflammatory response induced by hemodynamic changes preceding intracranial aneurysm formation: experimental study in rats

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7 January 2021

Loss and apoptosis of smooth muscle cells in intracranial aneurysms. Studies with in situ DNA end labeling and antibody against single-stranded DNA

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7 January 2021

10.1038/LABINVEST.2009.133

Identifiers

DOI

1 reference

16 March 2018

1 reference

16 March 2018