Respiratory syncytial virus activates epidermal growth factor receptor to suppress interferon regulatory factor 1-dependent interferon-lambda and antiviral defense in airway epithelium. (Q50143928)

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scientific article published on 7 February 2018

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English	Respiratory syncytial virus activates epidermal growth factor receptor to suppress interferon regulatory factor 1-dependent interferon-lambda and antiviral defense in airway epithelium.	scientific article published on 7 February 2018

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14 March 2020

Respiratory syncytial virus activates epidermal growth factor receptor to suppress interferon regulatory factor 1-dependent interferon-lambda and antiviral defense in airway epithelium (English)

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14 March 2020

author name string

A Kalinowski

1

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B T Galen

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I F Ueki

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Y Sun

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A Mulenos

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A Osafo-Addo

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B Clark

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J Joerns

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W Liu

9

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J A Nadel

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C S Dela Cruz

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J L Koff

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publication date

7 February 2018

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Mucosal Immunology

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11

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3

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958-967

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https://scigraph.springernature.com/pub.10.1038/mi.2017.120

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Respiratory syncytial virus infection in adults

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Activation of the epidermal growth factor receptor by respiratory syncytial virus results in increased inflammation and delayed apoptosis

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EGFR activation suppresses respiratory virus-induced IRF1-dependent CXCL10 production

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EGFR Interacts with the Fusion Protein of Respiratory Syncytial Virus Strain 2-20 and Mediates Infection and Mucin Expression

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Respiratory virus-induced EGFR activation suppresses IRF1-dependent interferon λ and antiviral defense in airway epithelium

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DDX60 Is Involved in RIG-I-Dependent and Independent Antiviral Responses, and Its Function Is Attenuated by Virus-Induced EGFR Activation

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Respiratory virus induction of alpha-, beta- and lambda-interferons in bronchial epithelial cells and peripheral blood mononuclear cells

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Lambda interferon renders epithelial cells of the respiratory and gastrointestinal tracts resistant to viral infections

1 reference

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Lambda interferon is the predominant interferon induced by influenza A virus infection in vivo

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Type-III interferon, not type-I, is the predominant interferon induced by respiratory viruses in nasal epithelial cells

1 reference

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7 January 2021

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Suppression of the induction of alpha, beta, and lambda interferons by the NS1 and NS2 proteins of human respiratory syncytial virus in human epithelial cells and macrophages [corrected]

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Requirement of a novel upstream response element in respiratory syncytial virus-induced IL-8 gene expression.

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Nonstructural proteins of respiratory syncytial virus suppress premature apoptosis by an NF-kappaB-dependent, interferon-independent mechanism and facilitate virus growth

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Peroxisomal MAVS activates IRF1-mediated IFN-λ production

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1 reference

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Respiratory syncytial virus infection of neonatal monocytes stimulates synthesis of interferon regulatory factor 1 and interleukin-1beta (IL-1beta)-converting enzyme and secretion of IL-1beta.

1 reference

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Immunity to RSV in Early-Life.

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Identifiers

10.1038/MI.2017.120

1 reference

Europe PubMed Central

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14 March 2020

1 reference

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14 March 2020

1 reference

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14 March 2020

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