Bcl-xL does not inhibit the function of Apaf-1. (Q48892500)

16 February 2018

title

Bcl-xL does not inhibit the function of Apaf-1. (English)

1 reference

16 February 2018

author

Ruth Kluck

3

1 reference

16 February 2018

author name string

Newmeyer DD

1

1 reference

16 February 2018

Bossy-Wetzel E

2

1 reference

16 February 2018

Wolf BB

4

1 reference

16 February 2018

Beere HM

5

1 reference

16 February 2018

Green DR

6

1 reference

16 February 2018

publication date

1 April 2000

1 reference

16 February 2018

Cell Death & Differentiation

published in

1 reference

16 February 2018

volume

7

1 reference

16 February 2018

page(s)

402-407

1 reference

16 February 2018

issue

4

1 reference

16 February 2018

https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400665

exact match

0 references

cites work

Caspases: enemies within

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Caspases and caspase inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

21 January 2018

Bcl-2 family proteins

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

The Bcl-2 protein family: arbiters of cell survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Genetics of programmed cell death in C. elegans: past, present and future.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cell-specific induction of apoptosis by microinjection of cytochrome c. Bcl-xL has activity independent of cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Apoptosis induced by microinjection of cytochrome c is caspase-dependent and is inhibited by Bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-2 regulates amplification of caspase activation by cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Human IAP-like protein regulates programmed cell death downstream of Bcl-xL and cytochrome c

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Caspases induce cytochrome c release from mitochondria by activating cytosolic factors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Autoactivation of procaspase-9 by Apaf-1-mediated oligomerization

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cell-free apoptosis in Xenopus egg extracts: inhibition by Bcl-2 and requirement for an organelle fraction enriched in mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cytochrome c activation of CPP32-like proteolysis plays a critical role in a Xenopus cell-free apoptosis system.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

CED-4 induces chromatin condensation in Schizosaccharomyces pombe and is inhibited by direct physical association with CED-9.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Interaction and regulation of the Caenorhabditis elegans death protease CED-3 by CED-4 and CED-9.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Interaction of CED-4 with CED-3 and CED-9: a molecular framework for cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-2 family members do not inhibit apoptosis by binding the caspase activator Apaf-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Apaf1 is required for mitochondrial pathways of apoptosis and brain development

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Inhibition of Bax channel-forming activity by Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Channel formation by antiapoptotic protein Bcl-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-2 family proteins as ion-channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Cytoprotection by Bcl-2 requires the pore-forming alpha5 and alpha6 helices

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-x(L) forms an ion channel in synthetic lipid membranes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

Checkpoints of dueling dimers foil death wishes

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

inferred from DOI database lookup

7 January 2021

based on heuristic

Cell-free reconstitution of Fas-, UV radiation- and ceramide-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400665

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.CDD.4400665

1 reference

16 February 2018

PubMed ID

10773825

1 reference

16 February 2018