Melatonin protects primary cultures of rat cortical neurones from NMDA excitotoxicity and hypoxia/reoxygenation. (Q48594289)

13 February 2018

title

Melatonin protects primary cultures of rat cortical neurones from NMDA excitotoxicity and hypoxia/reoxygenation. (English)

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13 February 2018

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Cazevieille C

1

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13 February 2018

Safa R

series ordinal

2

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13 February 2018

Osborne NN

series ordinal

3

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13 February 2018

language of work or name

English

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publication date

1 September 1997

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13 February 2018

number of pages

5

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based on heuristic

inferred from page(s)

published in

Brain Research

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13 February 2018

volume

768

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13 February 2018

issue

1-2

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13 February 2018

page(s)

120-124

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13 February 2018

Superoxide and nitric oxide cooperation in hypoxia/reoxygenation-induced neuron injury.

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Retinal neurones containing kainate receptors are influenced by exogenous kainate and ischaemia while neurones lacking these receptors are not -- melatonin counteracts the effects of ischaemia and kainate.

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Glutamate neurotoxicity in cortical cell culture is calcium dependent

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Melatonin protects primary cultures of cerebellar granule neurons from kainate but not from N-methyl-D-aspartate excitotoxicity

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NMDA-receptor activation increases cytoplasmic calcium concentration in cultured spinal cord neurones

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Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stress

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10.1016/S0006-8993(97)00611-2

7 January 2021

Identifiers

DOI

1 reference

13 February 2018

PubMed ID

9369308

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13 February 2018