Amyloid-beta: a (life) preserver for the brain. (Q48430388)

11 February 2018

title

Amyloid-beta: a (life) preserver for the brain. (English)

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11 February 2018

author

George Perry

4

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11 February 2018

Mark Anthony Smith

5

object named as

Mark A Smith

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11 February 2018

3

Craig S Atwood

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11 February 2018

Mark E. Obrenovich

1

object named as

Mark E Obrenovich

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11 February 2018

author name string

James A Joseph

2

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11 February 2018

publication date

1 November 2002

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11 February 2018

published in

Neurobiology of Aging

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11 February 2018

volume

23

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11 February 2018

issue

6

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11 February 2018

page(s)

1097-1099

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11 February 2018

Dramatic aggregation of Alzheimer abeta by Cu(II) is induced by conditions representing physiological acidosis.

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1 reference

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7 January 2021

Characterization of Copper Interactions with Alzheimer Amyloid β Peptides

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Neuroinflammatory Responses in the Alzheimer's Disease Brain Promote the Oxidative Post-translational Modification of Amyloid Deposits

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7 January 2021

GM1 inhibits amyloid beta-protein-induced cytokine release

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7 January 2021

Evidence that the beta-amyloid plaques of Alzheimer's disease represent the redox-silencing and entombment of abeta by zinc

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7 January 2021

Alzheimer's disease amyloid-beta binds copper and zinc to generate an allosterically ordered membrane-penetrating structure containing superoxide dismutase-like subunits

1 reference

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7 January 2021

Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer's disease

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7 January 2021

Copernicus revisited: amyloid beta in Alzheimer's disease

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7 January 2021

Amyloid-beta is an antioxidant for lipoproteins in cerebrospinal fluid and plasma

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7 January 2021

Hormone binding to natural mutants of human serum albumin

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7 January 2021

Abeta immunization: moving Abeta peptide from brain to blood

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Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

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7 January 2021

Cholesterol accumulates in senile plaques of Alzheimer disease patients and in transgenic APP(SW) mice

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7 January 2021

Neuronal oxidative stress precedes amyloid-beta deposition in Down syndrome

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Oxidative damage is the earliest event in Alzheimer disease

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7 January 2021

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7 January 2021

Redox-active iron mediates amyloid-beta toxicity

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7 January 2021

The state versus amyloid-beta: the trial of the most wanted criminal in Alzheimer disease

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In Situ Oxidative Catalysis by Neurofibrillary Tangles and Senile Plaques in Alzheimer’s Disease

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Toward a comprehensive theory for Alzheimer's disease. Hypothesis: Alzheimer's disease is caused by the cerebral accumulation and cytotoxicity of amyloid beta-protein.

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Alzheimer's disease results from the cerebral accumulation and cytotoxicity of amyloid beta-protein

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7 January 2021

Iron accumulation in Alzheimer disease is a source of redox-generated free radicals

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7 January 2021

Arson. Tracking the culprit in Alzheimer's disease

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7 January 2021

Amyloid-beta and tau serve antioxidant functions in the aging and Alzheimer brain

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7 January 2021

Induction of Alzheimer-like beta-amyloid immunoreactivity in the brains of rabbits with dietary cholesterol.

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7 January 2021

Intraneuronal beta-amyloid immunoreactivity in the CNS.

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7 January 2021

Amyloid beta protein enhances the survival of hippocampal neurons in vitro

1 reference

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7 January 2021

10.1016/S0197-4580(02)00038-6

Identifiers

DOI

1 reference

11 February 2018

PubMed ID

12470809

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11 February 2018