Residual substantia nigra neuromelanin in Parkinson's disease is cross-linked to alpha-synuclein. (Q48386115)

11 February 2018

title

Residual substantia nigra neuromelanin in Parkinson's disease is cross-linked to alpha-synuclein. (English)

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11 February 2018

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1

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Leonardo Lopiano

series ordinal

5

object named as

Leonardo Lopiano

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11 February 2018

Sabrina Giraudo

2

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11 February 2018

Silvia Coha

series ordinal

3

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11 February 2018

Bruno Bergamasco

series ordinal

4

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11 February 2018

publication date

1 June 2003

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11 February 2018

Neurochemistry International

published in

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11 February 2018

volume

42

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11 February 2018

page(s)

603-606

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11 February 2018

issue

7

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11 February 2018

NMR Studies of Melanins: Characterization of a Soluble Melanin Free Acid From Sepia Ink

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Evidence for a glycidic-lipidic matrix in human neuromelanin, potentially responsible for the enhanced iron sequestering ability of substantia nigra.

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Isolation and 13C-NMR characterization of an insoluble proteinaceous fraction from substantia nigra of patients with Parkinson's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Structure/function in neuroprotection and apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Parkin ubiquitinates the alpha-synuclein-interacting protein, synphilin-1: implications for Lewy-body formation in Parkinson disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Parkin and the molecular pathways of Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Enhanced vulnerability to oxidative stress by alpha-synuclein mutations and C-terminal truncation

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Parkinson's disease. First of two parts

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

1 reference

7 January 2021

Nuclear magnetic relaxation dispersion profiles of substantia nigra pars compacta in Parkinson's disease patients are consistent with protein aggregation.

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Proteasomal function is impaired in substantia nigra in Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Both familial Parkinson's disease mutations accelerate alpha-synuclein aggregation

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Etiology and pathogenesis of Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Mitochondrial involvement in Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Glutathione elevation and its protective role in acrolein-induced protein damage in synaptosomal membranes: relevance to brain lipid peroxidation in neurodegenerative disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Ubiquitination of a new form of alpha-synuclein by parkin from human brain: implications for Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodies

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

Expression of mutant alpha-synuclein causes increased susceptibility to dopamine toxicity

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

alpha-synuclein metabolism and aggregation is linked to ubiquitin-independent degradation by the proteasome

1 reference

https://api.crossref.org/works/10.1016%2FS0197-0186%2802%2900161-4

7 January 2021

10.1016/S0197-0186(02)00161-4

Identifiers

DOI

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11 February 2018

PubMed ID

12590943

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11 February 2018