Hydroxyl radical formation via iron-mediated Fenton chemistry is inhibited by methylated catechols. (Q48351614)

10 February 2018

title

Hydroxyl radical formation via iron-mediated Fenton chemistry is inhibited by methylated catechols. (English)

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10 February 2018

author name string

A J Nappi

series ordinal

1

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10 February 2018

E Vass

series ordinal

2

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10 February 2018

publication date

1 September 1998

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10 February 2018

Biochimica et Biophysica Acta

published in

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10 February 2018

volume

1425

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10 February 2018

issue

1

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10 February 2018

page(s)

159-167

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10 February 2018

Free radical mechanisms in the central nervous system: an overview

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

The oxidant stress hypothesis in Parkinson's disease: evidence supporting it

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Oxygen radicals as key mediators in neurological disease: fact or fiction?

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Reactive oxygen species and the central nervous system

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Oxygen, antioxidants and brain dysfunction

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Catecholamines enhance dihydrolipoamide dehydrogenase inactivation by the copper Fenton system. Enzyme protection by copper chelators

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Hydroxyl radical formation resulting from the interaction of nitric oxide and hydrogen peroxide

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

The roles of neuromelanin, binding of metal ions, and oxidative cytotoxicity in the pathogenesis of Parkinson's disease: a hypothesis

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

On the functional of neuromelanin.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Oxidative damage caused by free radicals produced during catecholamine autoxidation: protective effects of O-methylation and melatonin

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Inhibitory effect of melanin precursors on arachidonic acid peroxidation

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Nitric oxide-induced nitration of catecholamine neurotransmitters: A key to neuronal degeneration?

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Intracranial microdialysis of salicylic acid to detect hydroxyl radical generation through dopamine autooxidation in the caudate nucleus: effects of MPP+

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Hydroxyl free radical (.OH) formation reflected by salicylate hydroxylation and neuromelanin. In vivo markers for oxidant injury of nigral neurons

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

The effects of hydroxyl radical attack on dopa, dopamine, 6-hydroxydopa, and 6-hydroxydopamine

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Comparative studies of enhanced iron-mediated production of hydroxyl radical by glutathione, cysteine, ascorbic acid, and selected catechols

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Phospholipid peroxidation induced by the catechol-Fe3+(Cu2+) complex: a possible mechanism of nigrostriatal cell damage

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Tissue levels of S-adenosylmethionine in aging rats

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Relationship between tissue levels of S-adenosylmethionine, S-adenosylhomocysteine, and transmethylation reactions

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Parkinson's disease-like effects of S-adenosyl-L-methionine: effects of L-dopa

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Brain S-adenosylmethionine levels are severely decreased in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

Levels of L-methionine S-adenosyltransferase activity in erythrocytes and concentrations of S-adenosylmethionine and S-adenosylhomocysteine in whole blood of patients with Parkinson's disease.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

A rationale for monoamine oxidase inhibition as neuroprotective therapy for Parkinson's disease

1 reference

https://api.crossref.org/works/10.1016%2FS0304-4165%2898%2900062-2

7 January 2021

10.1016/S0304-4165(98)00062-2

Identifiers

DOI

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10 February 2018

PubMed ID

9813302

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10 February 2018