Fibroblast growth factor receptor 3 kinase domain mutation increases cortical progenitor proliferation via mitogen-activated protein kinase activation. (Q48332396)

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scientific article published on 20 December 2006

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English	Fibroblast growth factor receptor 3 kinase domain mutation increases cortical progenitor proliferation via mitogen-activated protein kinase activation.	scientific article published on 20 December 2006

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scholarly article

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

Fibroblast growth factor receptor 3 kinase domain mutation increases cortical progenitor proliferation via mitogen-activated protein kinase activation. (English)

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

3

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

author name string

Rachel E Thomson

1

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

Francesca Pellicano

2

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

language of work or name

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publication date

20 December 2006

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

Journal of Neurochemistry

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

100

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

6

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

1565-1578

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Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

Numbers, time and neocortical neuronogenesis: a general developmental and evolutionary model

1 reference

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Skeletal overgrowth and deafness in mice lacking fibroblast growth factor receptor 3.

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Spatial and temporal patterns of ERK signaling during mouse embryogenesis

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Coordinate expression of Fgf8, Otx2, Bmp4, and Shh in the rostral prosencephalon during development of the telencephalic and optic vesicles.

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Mechanisms underlying differential responses to FGF signaling

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Fibroblast growth factor receptor 3 is a negative regulator of bone growth

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Impaired cerebral cortex development and blood pressure regulation in FGF-2-deficient mice

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Cellular signaling by fibroblast growth factor receptors

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Tyrosine phosphorylation of Sprouty2 enhances its interaction with c-Cbl and is crucial for its function

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Fibroblast growth factors in the developing central nervous system

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Neocortex patterning by the secreted signaling molecule FGF8

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Molecular regionalization of the neocortex is disrupted in Fgf8 hypomorphic mutants

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Generating the cerebral cortical area map

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Sprouty: how does the branch manager work?

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hSpry2 is targeted to the ubiquitin-dependent proteasome pathway by c-Cbl

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Sprouty1 and Sprouty2 provide a control mechanism for the Ras/MAPK signalling pathway

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FGF signaling through FGFR1 is required for olfactory bulb morphogenesis.

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The cerebral cortex malformation in thanatophoric dysplasia: neuropathology and pathogenesis

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FGFR3 regulates brain size by controlling progenitor cell proliferation and apoptosis during embryonic development

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A neonatal lethal mutation in FGFR3 uncouples proliferation and differentiation of growth plate chondrocytes in embryos.

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Highly activated Fgfr3 with the K644M mutation causes prolonged survival in severe dwarf mice

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Modulation of signalling by Sprouty: a developing story

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Cloning of the mouse Sef gene and comparative analysis of its expression with Fgf8 and Spry2 during embryogenesis.

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Area identity shifts in the early cerebral cortex of Emx2-/- mutant mice.

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Sprouty proteins: multifaceted negative-feedback regulators of receptor tyrosine kinase signaling.

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Mechanisms of cerebral cortical patterning in mice and humans

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Emx2 and Pax6 control regionalization of the pre-neuronogenic cortical primordium.

1 reference

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Graded activation of fibroblast growth factor receptor 3 by mutations causing achondroplasia and thanatophoric dysplasia

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Phosphoinositide-specific phospholipase C and mitogenic signaling

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Fibroblast growth factor receptor 1 is required for the proliferation of hippocampal progenitor cells and for hippocampal growth in mouse.

1 reference

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Unique expression pattern of the FGF receptor 3 gene during mouse organogenesis

1 reference

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FGFR1 is required for the development of the auditory sensory epithelium

1 reference

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Fibroblast growth factors, their receptors and signaling

1 reference

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Fgfr3 expression by astrocytes and their precursors: evidence that astrocytes and oligodendrocytes originate in distinct neuroepithelial domains

1 reference

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Fibroblast growth factors and their receptors in the central nervous system

1 reference

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Sprouty fine-tunes EGF signaling through interlinked positive and negative feedback loops

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Extracellular signal-regulated kinases 1/2 are required for adult retinal ganglion cell axon regeneration induced by fibroblast growth factor-2.

1 reference

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Cell signaling by receptor tyrosine kinases

1 reference

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Sprouty2, a mouse deafness gene, regulates cell fate decisions in the auditory sensory epithelium by antagonizing FGF signaling

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Loss of glutamatergic pyramidal neurons in frontal and temporal cortex resulting from attenuation of FGFR1 signaling is associated with spontaneous hyperactivity in mice

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A novel skeletal dysplasia with developmental delay and acanthosis nigricans is caused by a Lys650Met mutation in the fibroblast growth factor receptor 3 gene

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FGFR1 is independently required in both developing mid- and hindbrain for sustained response to isthmic signals

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Promotion and attenuation of FGF signaling through the Ras-MAPK pathway

1 reference

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Changes in cerebral cortex size are governed by fibroblast growth factor during embryogenesis

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COUP-TFI: an intrinsic factor for early regionalization of the neocortex

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Identifiers

10.1111/J.1471-4159.2006.04285.X

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

1 reference

Europe PubMed Central

10 February 2018

http://europepmc.org/abstract/MED/17181553

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