Light and circadian rhythmicity regulate MAP kinase activation in the suprachiasmatic nuclei. (Q48239962)

9 February 2018

title

Light and circadian rhythmicity regulate MAP kinase activation in the suprachiasmatic nuclei. (English)

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9 February 2018

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Obrietan K

1

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9 February 2018

Impey S

series ordinal

2

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9 February 2018

Storm DR

series ordinal

3

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9 February 2018

language of work or name

English

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publication date

1 December 1998

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9 February 2018

published in

Nature Neuroscience

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9 February 2018

volume

1

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9 February 2018

page(s)

693-700

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9 February 2018

issue

8

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9 February 2018

https://scigraph.springernature.com/pub.10.1038/3695

exact match

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1 reference

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c-fos mRNA in the suprachiasmatic nuclei in vitro shows a circadian rhythm and responds to a serotonergic agonist

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A Functional analysis of circadian pacemakers in nocturnal rodents

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Light regulates expression of a Fos-related protein in rat suprachiasmatic nuclei

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Photic and circadian regulation of c-fos gene expression in the hamster suprachiasmatic nucleus.

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Light pulses that shift rhythms induce gene expression in the suprachiasmatic nucleus.

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Block of c-Fos and JunB expression by antisense oligonucleotides inhibits light-induced phase shifts of the mammalian circadian clock

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Regulation of CREB phosphorylation in the suprachiasmatic nucleus by light and a circadian clock

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NMDA receptor antagonists block the effects of light on circadian behavior in the mouse

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NMDA as well as non-NMDA receptor antagonists can prevent the phase-shifting effects of light on the circadian system of the golden hamster

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Membrane depolarization and calcium influx stimulate MEK and MAP kinase via activation of Ras.

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Calcium influx induces neurite growth through a Src-Ras signaling cassette

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Calcium activation of Ras mediated by neuronal exchange factor Ras-GRF.

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Stimulation of growth factor receptor signal transduction by activation of voltage-sensitive calcium channels

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Protein tyrosine kinase PYK2 involved in Ca(2+)-induced regulation of ion channel and MAP kinase functions

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A synaptic Ras-GTPase activating protein (p135 SynGAP) inhibited by CaM kinase II

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Regulation of transcription by MAP kinase cascades

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Phosphorylation of the MAP kinase ERK2 promotes its homodimerization and nuclear translocation

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Growth factors induce nuclear translocation of MAP kinases (p42mapk and p44mapk) but not of their activator MAP kinase kinase (p45mapkk) in fibroblasts

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Sustained activation of the mitogen-activated protein (MAP) kinase cascade may be required for differentiation of PC12 cells. Comparison of the effects of nerve growth factor and epidermal growth factor

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Cyclic AMP stimulates somatostatin gene transcription by phosphorylation of CREB at serine 133

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Nerve growth factor activates extracellular signal-regulated kinase and p38 mitogen-activated protein kinase pathways to stimulate CREB serine 133 phosphorylation

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Cross talk between ERK and PKA is required for Ca2+ stimulation of CREB-dependent transcription and ERK nuclear translocation.

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Microtubule-associated protein 2 kinases, ERK1 and ERK2, undergo autophosphorylation on both tyrosine and threonine residues: implications for their mechanism of activation

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Ca(2+)-dependent routes to Ras: mechanisms for neuronal survival, differentiation, and plasticity?

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Resetting the biological clock: mediation of nocturnal CREB phosphorylation via light, glutamate, and nitric oxide

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Resetting the biological clock: mediation of nocturnal circadian shifts by glutamate and NO

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Nitric oxide mediates N-methyl-D-aspartate receptor-induced activation of p21ras

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A differential response of two putative mammalian circadian regulators, mper1 and mper2, to light.

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Light-induced resetting of a mammalian circadian clock is associated with rapid induction of the mPer1 transcript.

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Three period homologs in mammals: differential light responses in the suprachiasmatic circadian clock and oscillating transcripts outside of brain.

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Closing the circadian loop: CLOCK-induced transcription of its own inhibitors per and tim.

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Developmentally regulated gene expression of all eight metabotropic glutamate receptors in hypothalamic suprachiasmatic and arcuate nuclei--a PCR analysis.

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In vivo measurement of a diurnal variation in vasopressin release in the rat suprachiasmatic nucleus

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Variation in the expression of the mRNA for protein kinase C isoforms in the rat suprachiasmatic nuclei, caudate putamen and cerebral cortex.

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RIGUI, a putative mammalian ortholog of the Drosophila period gene

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Circadian oscillation of a mammalian homologue of the Drosophila period gene

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Day-night variation of preprosomatostatin messenger RNA level in the suprachiasmatic nucleus.

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Induction of CRE-mediated gene expression by stimuli that generate long-lasting LTP in area CA1 of the hippocampus.

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7 January 2021

Identifiers

DOI

10.1038/3695

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9 February 2018

PubMed ID

10196585

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9 February 2018