Paradoxical decrease in mutant frequencies and chromosomal rearrangements in a transgenic lacZ reporter gene in Ku80 null mice deficient in DNA double strand break repair. (Q48215784)

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English	Paradoxical decrease in mutant frequencies and chromosomal rearrangements in a transgenic lacZ reporter gene in Ku80 null mice deficient in DNA double strand break repair.	scientific article

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scholarly article

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

Paradoxical decrease in mutant frequencies and chromosomal rearrangements in a transgenic lacZ reporter gene in Ku80 null mice deficient in DNA double strand break repair. (English)

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

author name string

Lynne D Rockwood

1

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

André Nussenzweig

2

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

Siegfried Janz

3

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

language of work or name

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publication date

1 August 2003

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

Mutation Research

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

529

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

1-2

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9 February 2018

http://europepmc.org/abstract/MED/12943919

51-58

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

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Sensing and repairing DNA double-strand breaks

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The nonhomologous DNA end joining pathway is important for chromosome stability in primary fibroblasts

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The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations

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Ku DNA end-binding protein modulates homologous repair of double-strand breaks in mammalian cells

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DNA ligase IV-deficient cells are more resistant to ionizing radiation in the absence of Ku70: Implications for DNA double-strand break repair

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Ku affects the ATM-dependent S phase checkpoint following ionizing radiation

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Homology-directed repair is a major double-strand break repair pathway in mammalian cells

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Saccharomyces cerevisiae Ku70 potentiates illegitimate DNA double-strand break repair and serves as a barrier to error-prone DNA repair pathways

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Different types of V(D)J recombination and end-joining defects in DNA double-strand break repair mutant mammalian cells

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Unrepaired DNA breaks in p53-deficient cells lead to oncogenic gene amplification subsequent to translocations.

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Oxygen metabolism causes chromosome breaks and is associated with the neuronal apoptosis observed in DNA double-strand break repair mutants

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Identifiers

10.1016/S0027-5107(03)00108-8

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/12943919

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