Attenuation of ischemia-induced cellular and behavioral deficits by X chromosome-linked inhibitor of apoptosis protein overexpression in the rat hippocampus. (Q48188556)

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English	Attenuation of ischemia-induced cellular and behavioral deficits by X chromosome-linked inhibitor of apoptosis protein overexpression in the rat hippocampus.	scientific article

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8 December 2019

Attenuation of ischemia-induced cellular and behavioral deficits by X chromosome-linked inhibitor of apoptosis protein overexpression in the rat hippocampus (English)

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8 December 2019

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D Xu

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Y Bureau

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D C McIntyre

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D W Nicholson

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P Liston

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Y Zhu

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W G Fong

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S J Crocker

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R G Korneluk

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G S Robertson

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1 June 1999

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8 December 2019

Journal of Neuroscience

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8 December 2019

19

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8 December 2019

5026-5033

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8 December 2019

NeuN, a neuronal specific nuclear protein in vertebrates

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Ischemia-induced neuronal apoptosis

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Induction of caspase-3-like protease may mediate delayed neuronal death in the hippocampus after transient cerebral ischemia.

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Caspases: killer proteases

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Distribution of neuronal apoptosis inhibitory protein-like immunoreactivity in the rat central nervous system

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https://pubmed.ncbi.nlm.nih.gov/10366635

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The gene for neuronal apoptosis inhibitory protein is partially deleted in individuals with spinal muscular atrophy

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Suppression of apoptosis in mammalian cells by NAIP and a related family of IAP genes

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Errors of homeostasis and deregulated apoptosis

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Immediate-early gene protein expression in neurons undergoing delayed death, but not necrosis, following hypoxic-ischaemic injury to the young rat brain

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Constitutive expression of zif268 in neocortex is regulated by synaptic activity

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Neuronal death in developmental models: possible implications in neuropathology

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Ischemia-induced CA1 neuronal death is preceded by elevated FosB and Jun expression and reduced NGFI-A and JunB levels.

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Activation and cleavage of caspase-3 in apoptosis induced by experimental cerebral ischemia.

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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The c-IAP-1 and c-IAP-2 proteins are direct inhibitors of specific caspases

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Neurobehavioral deficit due to ischemic brain damage limited to half of the CA1 sector of the hippocampus.

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Attenuation of delayed neuronal death after mild focal ischemia in mice by inhibition of the caspase family.

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Expression of inhibitor of apoptosis proteins (IAPs) in rat granulosa cells during ovarian follicular development and atresia

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https://pubmed.ncbi.nlm.nih.gov/10366635

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A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Cell death attenuation by 'Usurpin', a mammalian DED-caspase homologue that precludes caspase-8 recruitment and activation by the CD-95 (Fas, APO-1) receptor complex.

1 reference

https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury.

1 reference

https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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The four-vessel occlusion rat model: method for complete occlusion of vertebral arteries and control of collateral circulation

1 reference

https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Elevation of neuronal expression of NAIP reduces ischemic damage in the rat hippocampus

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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X-linked IAP is a direct inhibitor of cell-death proteases

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Mammalian cell death proteases: a family of highly conserved aspartate specific cysteine proteases

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Differential transcription and translation of immediate early genes in the gerbil hippocampus after transient global ischemia

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https://pubmed.ncbi.nlm.nih.gov/10366635

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Does neuronal damage of CA1 relate to spatial memory performance of rats subjected to transient forebrain ischemia?

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https://pubmed.ncbi.nlm.nih.gov/10366635

12 December 2020

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Identifiers

10.1523/JNEUROSCI.19-12-05026.1999

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8 December 2019

1 reference

Europe PubMed Central

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8 December 2019

1 reference

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8 December 2019

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