Adaptive depression in synaptic transmission in the nucleus of the solitary tract after in vivo chronic intermittent hypoxia: evidence for homeostatic plasticity. (Q48188039)

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English	Adaptive depression in synaptic transmission in the nucleus of the solitary tract after in vivo chronic intermittent hypoxia: evidence for homeostatic plasticity.	scientific article

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Adaptive depression in synaptic transmission in the nucleus of the solitary tract after in vivo chronic intermittent hypoxia: evidence for homeostatic plasticity (English)

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Angelina Ramirez-Navarro

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Diana L Kunze

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Journal of Neuroscience

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Presynaptic long-term depression at a central glutamatergic synapse: a role for CaMKII.

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Chronic hypoxia enhances the phrenic nerve response to arterial chemoreceptor stimulation in anesthetized rats

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Central nervous system mechanisms of ventilatory acclimatization to hypoxia

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Responses of single-unit carotid body chemoreceptors in adult rats

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Maintaining the stability of neural function: a homeostatic hypothesis

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Invited review: Physiological consequences of intermittent hypoxia: systemic blood pressure

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Reliability of monosynaptic sensory transmission in brain stem neurons in vitro.

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Enhanced spontaneous transmitter release is the earliest consequence of neocortical hypoxia that can explain the disruption of normal circuit function.

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Chronic intermittent hypoxia elicits serotonin-dependent plasticity in the central neural control of breathing.

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SNARE function analyzed in synaptobrevin/VAMP knockout mice

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Genetic ablation of the t-SNARE SNAP-25 distinguishes mechanisms of neuroexocytosis

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Short-term synaptic plasticity

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The molecular basis of CaMKII function in synaptic and behavioural memory

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Multiple mechanisms for the potentiation of AMPA receptor-mediated transmission by alpha-Ca2+/calmodulin-dependent protein kinase II.

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Ventilatory changes during intermittent hypoxia: importance of pattern and duration.

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Phosphorylation state, solubility, and activity of calcium/calmodulin-dependent protein kinase II alpha in transient focal ischemia in mouse brain.

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Progesterone reverses the neuronal responses to hypoxia in rat nucleus tractus solitarius in vitro

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Dopamine modulates synaptic transmission in the nucleus of the solitary tract

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Essential function of alpha-calcium/calmodulin-dependent protein kinase II in neurotransmitter release at a glutamatergic central synapse

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Chronic intermittent hypoxia enhances ventilatory long-term facilitation in awake rats.

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Induction of sensory long-term facilitation in the carotid body by intermittent hypoxia: implications for recurrent apneas

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Hypoxia-induced functional alterations in adult rat neocortex

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Characterization of calcium currents in aortic baroreceptor neurons

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Deletion of the synaptic protein interaction site of the N-type (CaV2.2) calcium channel inhibits secretion in mouse pheochromocytoma cells

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An isolated pool of vesicles recycles at rest and drives spontaneous neurotransmission

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1 reference

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

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https://pubmed.ncbi.nlm.nih.gov/17460079

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Inactivation and self-association of Ca2+/calmodulin-dependent protein kinase II during autophosphorylation.

1 reference

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based on heuristic

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1 reference

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1 reference

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based on heuristic

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Distinct synaptic loci of Ca2+/calmodulin-dependent protein kinase II necessary for long-term potentiation and depression

1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

12 December 2020

based on heuristic

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Inhibition of neurotransmission by peptides containing the synaptic protein interaction site of N-type Ca2+ channels

1 reference

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based on heuristic

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1 reference

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based on heuristic

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Afferent synaptic drive of rat medial nucleus tractus solitarius neurons: dynamic simulation of graded vesicular mobilization, release, and non-NMDA receptor kinetics.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

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based on heuristic

inferred from PubMed ID database lookup

Alteration of Ca2+ dependence of neurotransmitter release by disruption of Ca2+ channel/syntaxin interaction.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

12 December 2020

based on heuristic

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Phosphorylation of the synaptic protein interaction site on N-type calcium channels inhibits interactions with SNARE proteins.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

12 December 2020

based on heuristic

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Differences in Ca2+ channels governing generation of miniature and evoked excitatory synaptic currents in spinal laminae I and II.

1 reference

https://pubmed.ncbi.nlm.nih.gov/17460079

12 December 2020

based on heuristic

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Identifiers

10.1523/JNEUROSCI.4946-06.2007

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