Activity-dependent depression of GABAergic IPSCs in cultured hippocampal neurons. (Q48167078)

9 February 2018

title

Activity-dependent depression of GABAergic IPSCs in cultured hippocampal neurons. (English)

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9 February 2018

author name string

K Jensen

series ordinal

1

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9 February 2018

J D Lambert

series ordinal

2

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M S Jensen

series ordinal

3

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9 February 2018

language of work or name

English

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publication date

1 July 1999

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9 February 2018

Journal of Neurophysiology

published in

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volume

82

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page(s)

42-49

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issue

1

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9 February 2018

Enhancement of synaptic transmission by 4-aminopyridine in hippocampal slices of the rat

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Paired-pulse facilitation and depression at unitary synapses in rat hippocampus: quantal fluctuation affects subsequent release.

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Role of presynaptic L-type Ca2+ channels in GABAergic synaptic transmission in cultured hippocampal neurons.

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4‐Aminopyridine and low Ca2+ differentiate presynaptic inhibition mediated by neuropeptide Y, baclofen and 2‐chloroadenosine in rat hippocampal CA1 in vitro

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Temporally distinct mechanisms of use-dependent depression at inhibitory synapses in the rat hippocampus in vitro.

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Ca2+ imaging of CNS axons in culture indicates reliable coupling between single action potentials and distal functional release sites.

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How quickly can GABAA receptors open?

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GABAB autoreceptors mediate activity-dependent disinhibition and enhance signal transmission in the dentate gyrus.

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Depression of the fast IPSP underlies paired-pulse facilitation in area CA1 of the rat hippocampus

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High-frequency firing helps replenish the readily releasable pool of synaptic vesicles.

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The modulation of rat hippocampal synaptic conductances by baclofen and gamma-aminobutyric acid

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21 January 2018

Identifiers

DOI

10.1152/JN.1999.82.1.42

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9 February 2018

PubMed ID

10400933

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9 February 2018