Activation of astrocytes by CNTF induces metabolic plasticity and increases resistance to metabolic insults. (Q48115865)

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title

Activation of astrocytes by CNTF induces metabolic plasticity and increases resistance to metabolic insults (English)

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Emmanuel Brouillet

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Nicole Déglon

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Luc Pellerin

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Gilles Bonvento

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Marc Dhenain

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9

Philippe Hantraye

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6

Martine Guillermier

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3

Angélique Colin

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5

Thierry Delzescaux

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author name string

Karin Pierre

2

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language of work or name

English

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publication date

1 July 2007

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published in

Journal of Neuroscience

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volume

27

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issue

27

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page(s)

7094-7104

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De novo-synthesized ceramide signals apoptosis in astrocytes via extracellular signal-regulated kinase

1 January 2020

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Structural organization of the perivascular astrocyte endfeet and their relationship with the endothelial glucose transporter: a confocal microscopy study

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CNTF reverses obesity-induced insulin resistance by activating skeletal muscle AMPK.

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Palmitic and stearic fatty acids induce caspase-dependent and -independent cell death in nerve growth factor differentiated PC12 cells

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Glial glutamate transporters mediate a functional metabolic crosstalk between neurons and astrocytes in the mouse developing cortex

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Intrathecal delivery of CNTF using encapsulated genetically modified xenogeneic cells in amyotrophic lateral sclerosis patients

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Neuroprotective effect of a CNTF-expressing lentiviral vector in the quinolinic acid rat model of Huntington's disease.

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Neuroprotective gene therapy for Huntington's disease using a polymer encapsulated BHK cell line engineered to secrete human CNTF.

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The AMP-activated protein kinase is involved in the regulation of ketone body production by astrocytes.

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Decreased metabolic response to visual stimulation in the superior colliculus of mice lacking the glial glutamate transporter GLT-1

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Glutamate uptake into astrocytes stimulates aerobic glycolysis: a mechanism coupling neuronal activity to glucose utilization

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Capacity for substrate utilization in oxidative metabolism by neurons, astrocytes, and oligodendrocytes from developing brain in primary culture.

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Ciliary neurotrophic factor prevents acute lipid-induced insulin resistance by attenuating ceramide accumulation and phosphorylation of c-Jun N-terminal kinase in peripheral tissues

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Quantitative histochemical changes in enzymes involved in energy metabolism in the rat brain during postnatal development. II. Glucose-6-phosphate dehydrogenase and β-hydroxybutyrate dehydrogenase

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Ultrastructural study of enzymes in reactive astrocytes: Clarification of astrocytic activity

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Corticostriatopallidal neuroprotection by adenovirus-mediated ciliary neurotrophic factor gene transfer in a rat model of progressive striatal degeneration

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Fatty acid oxidation and ketogenesis by astrocytes in primary culture

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Dehydrogenase activity in reactive and neoplastic astrocytes.

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beta-Hydroxybutyrate fuels synaptic function during development. Histological and physiological evidence in rat hippocampal slices

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Ciliary neurotrophic factor improves diabetic parameters and hepatic steatosis and increases basal metabolic rate in db/db mice

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Cell-specific localization of monocarboxylate transporters, MCT1 and MCT2, in the adult mouse brain revealed by double immunohistochemical labeling and confocal microscopy

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D-beta-hydroxybutyrate rescues mitochondrial respiration and mitigates features of Parkinson disease

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Role of carnitine palmitoyltransferase I in the control of ketogenesis in primary cultures of rat astrocytes.

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Ciliary neurotrophic factor activates astrocytes, redistributes their glutamate transporters GLAST and GLT-1 to raft microdomains, and improves glutamate handling in vivo.

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Is there an astrocyte-neuron ketone body shuttle?

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Basal levels of metabolic activity are elevated in Genetic Absence Epilepsy Rats from Strasbourg (GAERS): measurement of regional activity of cytochrome oxidase and lactate dehydrogenase by histochemistry

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Ischemic cell death in brain neurons

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12 December 2020

10.1523/JNEUROSCI.0174-07.2007

Identifiers

DOI

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